Circadian dysfunction in response to in vivo treatment with the mitochondrial toxin 3-nitropropionic acid. - Wikidata - awgldk - TriplyDB

Circadian dysfunction in response to in vivo treatment with the mitochondrial toxin 3-nitropropionic acid.

Circadian dysfunction in response to in vivo treatment with the mitochondrial toxin 3-nitropropionic acid.

http://www.wikidata.org/entity/Q37476957

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'The clocks that time us'--circadian rhythms in neurodegenerative disorders The Circadian Clock Gene Period1 Connects the Molecular Clock to Neural Activity in the Suprachiasmatic Nucleus.The Circadian Nature of Mitochondrial Biology.

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Circadian dysfunction in response to in vivo treatment with the mitochondrial toxin 3-nitropropionic acid.

http://www.wikidata.org/entity/Q37476957

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 13 January 2014

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Circadian dysfunction in respo ...... l toxin 3-nitropropionic acid.

@en

Circadian dysfunction in respo ...... l toxin 3-nitropropionic acid.

@nl

type

label

Circadian dysfunction in respo ...... l toxin 3-nitropropionic acid.

@en

Circadian dysfunction in respo ...... l toxin 3-nitropropionic acid.

@nl

prefLabel

Circadian dysfunction in respo ...... l toxin 3-nitropropionic acid.

@en

Circadian dysfunction in respo ...... l toxin 3-nitropropionic acid.

@nl

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Circadian dysfunction in respo ...... l toxin 3-nitropropionic acid.

@en

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Christopher S Colwell

http://www.w3.org/2001/XMLSchema#string

Dawn H Loh

http://www.w3.org/2001/XMLSchema#string

Elizabeth Hernández-Echeagaray

http://www.w3.org/2001/XMLSchema#string

Yu Tahara

http://www.w3.org/2001/XMLSchema#string

P2860

PERIOD2::LUCIFERASE real-time reporting of circadian dynamics reveals persistent circadian oscillations in mouse peripheral tissues.

Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

Mitochondrial dysfunction and oxidative stress in Parkinson's disease

Striatal glucose metabolism and dopamine D2 receptor binding in asymptomatic gene carriers and patients with Huntington's disease

Circadian dysfunction may be a key component of the non-motor symptoms of Parkinson's disease: insights from a transgenic mouse model

The genetics of mammalian circadian order and disorder: implications for physiology and disease

Mitochondrial fission, fusion, and stress

Striatal glucose metabolism and dopamine D2 receptor binding in asymptomatic gene carriers and patients with Huntington's disease

Linking neural activity and molecular oscillations in the SCN.

N-terminal mutant huntingtin associates with mitochondria and impairs mitochondrial trafficking.

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e00133

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scholarly article

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10.1042/AN20130042

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1

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6

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2014-01-13T00:00:00Z

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24328694

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circadian rhythm

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3891360

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