Contrasting roles of the IL-1 and IL-18 receptors in MyD88-dependent contact hypersensitivity.
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IL-18 and Cutaneous Inflammatory DiseasesMechanisms of chemical-induced innate immunity in allergic contact dermatitis.Contact sensitizers induce skin inflammation via ROS production and hyaluronic acid degradationInsulin-like growth factor-1 stimulates regulatory T cells and suppresses autoimmune disease.IL-1 receptor signaling is required at multiple stages of sensitization and elicitation of the contact hypersensitivity responseLarge increases in adipose triacylglycerol flux in Cushingoid CRH-Tg mice are explained by futile cyclingDanger, intracellular signaling, and the orchestration of dendritic cell function in skin sensitization.The involvement of the Toll-like receptor signaling and Nrf2-Keap1 pathways in the in vitro regulation of IL-8 and HMOX1 for skin sensitization.Requirement of MyD88 signaling in keratinocytes for Langerhans cell migration and initiation of atopic dermatitis-like symptoms in mice.Lack of the purinergic receptor P2X(7) results in resistance to contact hypersensitivityLangerhans cells require MyD88-dependent signals for Candida albicans response but not for contact hypersensitivity or migrationAn ITAM-Syk-CARD9 signalling axis triggers contact hypersensitivity by stimulating IL-1 production in dendritic cells.Antimicrobial Peptide Epinecidin-1 Modulates MyD88 Protein Levels via the Proteasome Degradation Pathway.1-Fluoro-2,4-dinitrobenzene and its derivatives act as secretagogues on rodent mast cells.Epicutaneous exposure to nickel induces nickel allergy in mice via a MyD88-dependent and interleukin-1-dependent pathwayNatural Killer Cells: Development, Maturation, and Clinical Utilization
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Contrasting roles of the IL-1 and IL-18 receptors in MyD88-dependent contact hypersensitivity.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on January 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Contrasting roles of the IL-1 ...... dent contact hypersensitivity.
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Contrasting roles of the IL-1 ...... dent contact hypersensitivity.
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Contrasting roles of the IL-1 ...... dent contact hypersensitivity.
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Contrasting roles of the IL-1 ...... dent contact hypersensitivity.
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Contrasting roles of the IL-1 ...... dent contact hypersensitivity.
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Contrasting roles of the IL-1 ...... dent contact hypersensitivity.
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Contrasting roles of the IL-1 ...... dent contact hypersensitivity.
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Liping Yang
Paul A Klekotka
Wayne M Yokoyama
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10.1038/JID.2009.242
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2010-01-01T00:00:00Z