MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation - Wikidata - awgldk - TriplyDB

MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation

MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation

http://www.wikidata.org/entity/Q37485846

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PARP-10, a novel Myc-interacting protein with poly(ADP-ribose) polymerase activity, inhibits transformation PTEN represses RNA Polymerase I transcription by disrupting the SL1 complex The RNA polymerase I transcription machinery Centromere RNA is a key component for the assembly of nucleoproteins at the nucleolus and centromere Emerging Roles for Maf1 beyond the Regulation of RNA Polymerase III Activity Nuclear PI3K signaling in cell growth and tumorigenesis LAS1L interacts with the mammalian Rix1 complex to regulate ribosome biogenesis Nucleolar stress is an early response to myocardial damage involving nucleolar proteins nucleostemin and nucleophosmin UBF levels determine the number of active ribosomal RNA genes in mammals Inhibition of RNA polymerase I as a therapeutic strategy to promote cancer-specific activation of p53 Identification of functional networks of estrogen- and c-Myc-responsive genes and their relationship to response to tamoxifen therapy in breast cancer.Controlling the Master: Chromatin Dynamics at the MYC Promoter Integrate Developmental Signaling Structure and epigenetics of nucleoli in comparison with non-nucleolar compartments The pre-rRNA processing factor DEF is rate limiting for the pathogenesis of MYCN-driven neuroblastoma.Mitotic occupancy and lineage-specific transcriptional control of rRNA genes by Runx2.A novel role for the Pol I transcription factor UBTF in maintaining genome stability through the regulation of highly transcribed Pol II genes.The HBx oncoprotein of hepatitis B virus potentiates cell transformation by inducing c-Myc-dependent expression of the RNA polymerase I transcription factor UBF Direct regulation of rRNA transcription by fibroblast growth factor 2 Ribosomal protein S27-like is a physiological regulator of p53 that suppresses genomic instability and tumorigenesis Nucleolar stress with and without p53.Inflammatory disease and lymphomagenesis caused by deletion of the Myc antagonist Mnt in T cells.Dysregulation of RNA polymerase I transcription during disease.c-MYC coordinately regulates ribosomal gene chromatin remodeling and Pol I availability during granulocyte differentiation PNRC accumulates in the nucleolus by interaction with B23/nucleophosmin via its nucleolar localization sequence Tcof1/Treacle is required for neural crest cell formation and proliferation deficiencies that cause craniofacial abnormalities.The role of Myc-induced protein synthesis in cancer.Identification of AMP-activated protein kinase targets by a consensus sequence search of the proteome.Downregulation of rRNA transcription triggers cell differentiation.RNA polymerase I activity is regulated at multiple steps in the transcription cycle: recent insights into factors that influence transcription elongation.Genome wide mapping of UBF binding-sites in mouse and human cell lines.Drosophila Low Temperature Viability Protein 1 (LTV1) Is Required for Ribosome Biogenesis and Cell Growth Downstream of Drosophila Myc (dMyc)RNA-polymerase-I-directed rDNA transcription, life and works.Depletion of NEAT1 lncRNA attenuates nucleolar stress by releasing sequestered P54nrb and PSF to facilitate c-Myc translation.Analysis of histone modifications at human ribosomal DNA in liver cancer cell Depletion of the cisplatin targeted HMGB-box factor UBF selectively induces p53-independent apoptotic death in transformed cells.Drosophila growth and development in the absence of dMyc and dMnt.Targeting MYC in cancer therapy: RNA processing offers new opportunities.Genomic characterization of the mouse ribosomal DNA locus The Potential of Targeting Ribosome Biogenesis in High-Grade Serous Ovarian Cancer.Therapeutic Approaches Targeting MYC-Driven Prostate Cancer

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MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation

http://www.wikidata.org/entity/Q37485846

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 29 July 2004

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation

@en

MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation.

@nl

type

label

MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation

@en

MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation.

@nl

prefLabel

MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation

@en

MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation.

@nl

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SJ.EMBOJ.7600335

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The EMBO Journal

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MAD1 and c-MYC regulate UBF and rDNA transcription during granulocyte differentiation

@en

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Anna Jenkins

http://www.w3.org/2001/XMLSchema#string

Hayley Snelling

http://www.w3.org/2001/XMLSchema#string

Katherine M Hannan

http://www.w3.org/2001/XMLSchema#string

Kerith Sharkey

http://www.w3.org/2001/XMLSchema#string

Manuela Palatsides

http://www.w3.org/2001/XMLSchema#string

Meaghan Wall

http://www.w3.org/2001/XMLSchema#string

Richard B Pearson

http://www.w3.org/2001/XMLSchema#string

Yves Brandenburger

http://www.w3.org/2001/XMLSchema#string

P2860

Systematic identification of pathways that couple cell growth and division in yeast.

G1 events and regulation of cell proliferation

N-myc enhances the expression of a large set of genes functioning in ribosome biogenesis and protein synthesis.

The MYC protein activates transcription of the alpha-prothymosin gene.

Myc induces the nucleolin and BN51 genes: possible implications in ribosome biogenesis

Ribosomal S6 kinase signaling and the control of translation.

Targeted disruption of the MYC antagonist MAD1 inhibits cell cycle exit during granulocyte differentiation.

Overlapping functions of the pRb family in the regulation of rRNA synthesis

MAD1 and p27(KIP1) cooperate to promote terminal differentiation of granulocytes and to inhibit Myc expression and cyclin E-CDK2 activity

Deconstructing myc.

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3325-3335

http://www.w3.org/2001/XMLSchema#string

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scholarly article

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10.1038/SJ.EMBOJ.7600335

http://www.w3.org/2001/XMLSchema#string

P407

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16

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23

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Gretchen Poortinga

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2004-07-29T00:00:00Z

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8426484

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15282543

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514509

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