Ablation of Perk in Schwann Cells Improves Myelination in the S63del Charcot-Marie-Tooth 1B Mouse.

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ER stress and the unfolded protein response in neurodegeneration.Endoplasmic Reticulum Protein Quality Control Failure in Myelin Disorders.Impairment of protein degradation and proteasome function in hereditary neuropathies.

P2860

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P2860

Ablation of Perk in Schwann Cells Improves Myelination in the S63del Charcot-Marie-Tooth 1B Mouse.

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http://www.wikidata.org/entity/Q37490449

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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2016年學術文章

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name

Ablation of Perk in Schwann Ce ...... Charcot-Marie-Tooth 1B Mouse.

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Ablation of Perk in Schwann Ce ...... Charcot-Marie-Tooth 1B Mouse.

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type

Item

label

Ablation of Perk in Schwann Ce ...... Charcot-Marie-Tooth 1B Mouse.

@en

Ablation of Perk in Schwann Ce ...... Charcot-Marie-Tooth 1B Mouse.

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prefLabel

Ablation of Perk in Schwann Ce ...... Charcot-Marie-Tooth 1B Mouse.

@en

Ablation of Perk in Schwann Ce ...... Charcot-Marie-Tooth 1B Mouse.

@nl

P1476

Ablation of Perk in Schwann Ce ...... l Charcot-Marie-Tooth 1B Mouse

@en

P2093

Douglas R Cavener

http://www.w3.org/2001/XMLSchema#string

M Laura Feltri

http://www.w3.org/2001/XMLSchema#string

Maurizio D'Antonio

http://www.w3.org/2001/XMLSchema#string

Nicolò Musner

http://www.w3.org/2001/XMLSchema#string

P2860

Mammalian transcription factor ATF6 is synthesized as a transmembrane protein and activated by proteolysis in response to endoplasmic reticulum stress

XBP1 mRNA is induced by ATF6 and spliced by IRE1 in response to ER stress to produce a highly active transcription factor

Deletion of the serine 34 codon from the major peripheral myelin protein P0 gene in Charcot-Marie-Tooth disease type 1B

Distinct roles of activating transcription factor 6 (ATF6) and double-stranded RNA-activated protein kinase-like endoplasmic reticulum kinase (PERK) in transcription during the mammalian unfolded protein response

Sustained translational repression by eIF2α-P mediates prion neurodegeneration

Nrf2 is a direct PERK substrate and effector of PERK-dependent cell survival

A regulatory link between ER-associated protein degradation and the unfolded-protein response.

Functional and genomic analyses reveal an essential coordination between the unfolded protein response and ER-associated degradation

Perk is essential for translational regulation and cell survival during the unfolded protein response

Translational control is required for the unfolded protein response and in vivo glucose homeostasis

P304

11350-11361

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scholarly article

P356

10.1523/JNEUROSCI.1637-16.2016

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English

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2016-11-01T00:00:00Z

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27807175

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P932

5148247

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Ablation of Perk in Schwann Cells Improves Myelination in the S63del Charcot-Marie-Tooth 1B Mouse.

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P2860

P2860

Ablation of Perk in Schwann Cells Improves Myelination in the S63del Charcot-Marie-Tooth 1B Mouse.

description

name

type

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P1433

P1476

P2093

P2860

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P356

P407

P433

P478

P50

P577

P698

P932

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P1433

P1476

P2093

P2860

P304

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P407

P433

P478

P50

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P698

P932