Improvement of Heart Failure by Human Amniotic Mesenchymal Stromal Cell Transplantation in Rats. - Wikidata - awgldk - TriplyDB

Improvement of Heart Failure by Human Amniotic Mesenchymal Stromal Cell Transplantation in Rats.

Improvement of Heart Failure by Human Amniotic Mesenchymal Stromal Cell Transplantation in Rats.

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Improvement of Heart Failure by Human Amniotic Mesenchymal Stromal Cell Transplantation in Rats.

http://www.wikidata.org/entity/Q37490948

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on July 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Improvement of Heart Failure b ...... Cell Transplantation in Rats.

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Improvement of Heart Failure b ...... Cell Transplantation in Rats.

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Improvement of Heart Failure b ...... Cell Transplantation in Rats.

@en

Improvement of Heart Failure b ...... Cell Transplantation in Rats.

@nl

prefLabel

Improvement of Heart Failure b ...... Cell Transplantation in Rats.

@en

Improvement of Heart Failure b ...... Cell Transplantation in Rats.

@nl

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The journal of Tehran Heart Center

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Improvement of Heart Failure b ...... Cell Transplantation in Rats.

@en

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Amir Darbandi Azar

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Elham Kalantari

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Mahdieh Faghihi

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Maliheh Nobakht

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Mohammad Molazem

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Nahid Aboutaleb

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Seyed Mohammad Taghi Razavi Tousi

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P2860

Emerging Novel Therapies for Heart Failure

Autologous transplantation of bone marrow cells improves damaged heart function.

Term Amniotic membrane is a high throughput source for multipotent Mesenchymal Stem Cells with the ability to differentiate into endothelial cells in vitro.

Transplanted human amniotic membrane-derived mesenchymal stem cells ameliorate carbon tetrachloride-induced liver cirrhosis in mouse.

Survival and development of neonatal rat cardiomyocytes transplanted into adult myocardium.

Heart disease and stroke statistics--2007 update: a report from the American Heart Association Statistics Committee and Stroke Statistics Subcommittee.

Caveolin-3 Overexpression Attenuates Cardiac Hypertrophy via Inhibition of T-type Ca2+ Current Modulated by Protein Kinase Cα in Cardiomyocytes

Relevance of matrix metalloproteinases and their inhibitors after myocardial infarction: a temporal and spatial window.

Bioengineered myocardium derived from induced pluripotent stem cells improves cardiac function and attenuates cardiac remodeling following chronic myocardial infarction in rats.

Mesenchymal stem cell transplantation improves regional cardiac remodeling following ovine infarction

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123-138

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scholarly article

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2016-07-01T00:00:00Z

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27956912

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5148815

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