Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways. - Wikidata - awgldk - TriplyDB

Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.

Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.

http://www.wikidata.org/entity/Q37519522

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Paradoxical role of CBX8 in proliferation and metastasis of colorectal cancer.RAS protein activator-like 1 is functionally involved in hypoxia resistance in breast cancer cells by targeting hypoxia inducible factor-1α.

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Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.

http://www.wikidata.org/entity/Q37519522

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 07 January 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.

@en

Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.

@nl

type

label

Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.

@en

Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.

@nl

prefLabel

Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.

@en

Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways.

@nl

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PNAS.1322484111

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Proceedings of the National Academy of Sciences of the United States of America

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Hypoxia promotes tumor cell motility via RhoA and ROCK1 signaling pathways

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Ann F Chambers

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P2860

Antiangiogenic therapy elicits malignant progression of tumors to increased local invasion and distant metastasis

Hypoxia-inducible factors mediate coordinated RhoA-ROCK1 expression and signaling in breast cancer cells

Accelerated metastasis after short-term treatment with a potent inhibitor of tumor angiogenesis

Targeting hypoxia in cancer therapy

The impact of O2 availability on human cancer

Heterozygous deficiency of PHD2 restores tumor oxygenation and inhibits metastasis via endothelial normalization.

Heterogeneous vascular dependence of tumor cell populations

Tumor vasculature is regulated by PHD2-mediated angiogenesis and bone marrow-derived cell recruitment.

Extracellular matrix-dependent tissue-specific gene expression in mammary epithelial cells requires both physical and biochemical signal transduction.

Vascular permeability in cancer and infection as related to macromolecular drug delivery, with emphasis on the EPR effect for tumor-selective drug targeting.

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887-888

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scholarly article

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10.1073/PNAS.1322484111

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