Treatment of mice with the neutrophil-depleting antibody RB6-8C5 results in early development of experimental lyme arthritis via the recruitment of Gr-1- polymorphonuclear leukocyte-like cells.
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Ly6 family proteins in neutrophil biologyThe innate interferon gamma response of BALB/c and C57BL/6 mice to in vitro Burkholderia pseudomallei infection.Potential role of decoy B7-H4 in the pathogenesis of rheumatoid arthritis: a mouse model informed by clinical dataCheating by type 3 secretion system-negative Pseudomonas aeruginosa during pulmonary infection.Different patterns of expression and of IL-10 modulation of inflammatory mediators from macrophages of Lyme disease-resistant and -susceptible mice.A paradoxical role for neutrophils in the pathogenesis of West Nile virusCD14 signaling reciprocally controls collagen deposition and turnover to regulate the development of lyme arthritis.Chemokine receptor CXCR2 mediates bacterial clearance rather than neutrophil recruitment in a murine model of pneumonic plagueInfiltrating neutrophils mediate the initial angiogenic switch in a mouse model of multistage carcinogenesis.Pyruvate protects pathogenic spirochetes from H2O2 killing.Bb2Bb3 regulation of murine Lyme arthritis is distinct from Ncf1 and independent of the phagocyte nicotinamide adenine dinucleotide phosphate oxidaseTopical flagellin protects the injured corneas from Pseudomonas aeruginosa infection.Infection of Interleukin 17 Receptor A-Deficient C3H Mice with Borrelia burgdorferi Does Not Affect Their Development of Lyme Arthritis and Carditis.Hyperglycemia Impairs Neutrophil-Mediated Bacterial Clearance in Mice Infected with the Lyme Disease PathogenOuter surface protein OspC is an antiphagocytic factor that protects Borrelia burgdorferi from phagocytosis by macrophages.Viable Borrelia burgdorferi enhances interleukin-10 production and suppresses activation of murine macrophages.Novel microbial virulence factor triggers murine lyme arthritis.Gene expression profiling provides insights into the pathways involved in inflammatory arthritis development: murine model of Lyme disease.Lyme arthritis: current concepts and a change in paradigm.CXCR2 Mediates Brucella-Induced Arthritis in Interferon γ-Deficient MiceAlternatively activated macrophages in infection and autoimmunity.Interactions between host immune response and antigenic variation that control Borrelia burgdorferi population dynamics.Presence of Arp specifically contributes to joint tissue edema associated with early-onset Lyme arthritis.Recruitment of macrophages and polymorphonuclear leukocytes in Lyme carditis.Gr-1 antibody induces STAT signaling, macrophage marker expression and abrogation of myeloid-derived suppressor cell activity in BM cells.Characterization of Stress and Innate Immunity Resistance of Wild-type and Δp66 Borrelia burgdorferi.
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Treatment of mice with the neutrophil-depleting antibody RB6-8C5 results in early development of experimental lyme arthritis via the recruitment of Gr-1- polymorphonuclear leukocyte-like cells.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on September 2004
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Treatment of mice with the neu ...... onuclear leukocyte-like cells.
@en
Treatment of mice with the neu ...... onuclear leukocyte-like cells.
@nl
type
label
Treatment of mice with the neu ...... onuclear leukocyte-like cells.
@en
Treatment of mice with the neu ...... onuclear leukocyte-like cells.
@nl
prefLabel
Treatment of mice with the neu ...... onuclear leukocyte-like cells.
@en
Treatment of mice with the neu ...... onuclear leukocyte-like cells.
@nl
P2093
P2860
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Treatment of mice with the neu ...... onuclear leukocyte-like cells.
@en
P2093
Charles R Brown
Christie M Loiacono
Victoria A Blaho
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P304
P356
10.1128/IAI.72.9.4956-4965.2004
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P577
2004-09-01T00:00:00Z