Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy - Wikidata - awgldk - TriplyDB

Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy

Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy

http://www.wikidata.org/entity/Q37524085

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Macrophage Phenotype in Kidney Injury and Repair Autophagy, Innate Immunity and Tissue Repair in Acute Kidney Injury Recent advances in understanding of chronic kidney disease Paradigm Shifts in Atherosclerotic Renovascular Disease: Where Are We Now?Interferon-regulatory factors determine macrophage phenotype polarization Dendritic cells and macrophages in the kidney: a spectrum of good and evil Macrophage in chronic kidney disease Renal Arterial Disease and Hypertension.Both PD-1 ligands protect the kidney from ischemia reperfusion injury.Therapeutic translation in acute kidney injury: the epithelial/endothelial axis Improved renal recovery in patients with atypical hemolytic uremic syndrome following rapid initiation of eculizumab treatment.Oxalate-induced chronic kidney disease with its uremic and cardiovascular complications in C57BL/6 mice.IRAK-M promotes alternative macrophage activation and fibroproliferation in bleomycin-induced lung injury.Acute kidney injury in 2014: a step towards understanding mechanisms of renal repair.Specific macrophage subtypes influence the progression of rhabdomyolysis-induced kidney injury An update on the role of the inflammasomes in the pathogenesis of kidney diseases.Optimizing Mouse Surgery with Online Rectal Temperature Monitoring and Preoperative Heat Supply. Effects on Post-Ischemic Acute Kidney Injury Heme Oxygenase-1 Regulates Myeloid Cell Trafficking in AKI IL-34 mediates acute kidney injury and worsens subsequent chronic kidney disease.Relationship of clusterin with renal inflammation and fibrosis after the recovery phase of ischemia-reperfusion injury Biliverdin Reductase A (BVRA) Mediates Macrophage Expression of Interleukin-10 in Injured Kidney Clinicopathological significance of CD206-positive macrophages in patients with acute tubulointerstitial disease.Heterogeneous susceptibility for uraemic media calcification and concomitant inflammation within the arterial tree.Renal artery stenosis: if and when to intervene Elevated BP after AKI.Management of atherosclerotic renovascular disease after Cardiovascular Outcomes in Renal Atherosclerotic Lesions (CORAL)Regulated necrosis-related molecule mRNA expression in humans and mice and in murine acute tissue injury and systemic autoimmunity leading to progressive organ damage, and progressive fibrosis.NLRP3 Deficiency Attenuates Renal Fibrosis and Ameliorates Mitochondrial Dysfunction in a Mouse Unilateral Ureteral Obstruction Model of Chronic Kidney Disease.Interaction between systemic inflammation and renal tubular epithelial cells.Immune system modulation of kidney regeneration--mechanisms and implications.Immune cells in experimental acute kidney injury.Mechanisms of maladaptive repair after AKI leading to accelerated kidney ageing and CKD.Necroinflammation in Kidney Disease.HIF-1α induction during reperfusion avoids maladaptive repair after renal ischemia/reperfusion involving miR127-3p.Association between Monocyte Count and Risk of Incident CKD and Progression to ESRD.Tumor necrosis factor superfamily ligand mRNA expression profiles differ between humans and mice during homeostasis and between various murine kidney injuries.Macrophage dynamics in AKI to CKD progression.Myeloid Cell HO-ming in AKI.Gene expression profiling of the Notch-AhR-IL22 axis at homeostasis and in response to tissue injury.Antithrombin III prevents progression of chronic kidney disease following experimental ischaemic-reperfusion injury.

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Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy

http://www.wikidata.org/entity/Q37524085

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 05 December 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy

@en

Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy.

@nl

type

label

Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy

@en

Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy.

@nl

altLabel

Macrophage Phenotype Controls Long-Term AKI Outcomes—Kidney Regeneration versus Atrophy

@en

prefLabel

Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy

@en

Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy.

@nl

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Journal of the American Society of Nephrology

P1476

Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy

@en

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Georg Lorenz

http://www.w3.org/2001/XMLSchema#string

Heni Eka Susanti

http://www.w3.org/2001/XMLSchema#string

Ingo Hartter

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Koichi S Kobayashi

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Maciej Lech

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Mi Ryu

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Regina Gröbmayr

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Richard A Flavell

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Shrikant R Mulay

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P2860

Tumor cells deactivate human monocytes by up-regulating IL-1 receptor associated kinase-M expression via CD44 and TLR4

IRAK-M is a negative regulator of Toll-like receptor signaling

Does AKI truly lead to CKD?

Pattern recognition receptors and inflammation

Severe impairment of interleukin-1 and Toll-like receptor signalling in mice lacking IRAK-4

Induction of IRAK-M is associated with lipopolysaccharide tolerance in a human endotoxemia model

CSF-1 signaling mediates recovery from acute kidney injury.

The chemokine system in diverse forms of macrophage activation and polarization

Wound repair and regeneration

Mechanisms of fibrosis: therapeutic translation for fibrotic disease

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292-304

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scholarly article

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10.1681/ASN.2013020152

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2

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25

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Hans-Joachim Anders

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2013-12-05T00:00:00Z

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24309188

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3904561

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