UNC5B receptor deletion exacerbates tissue injury in response to AKI. - Wikidata - awgldk - TriplyDB

UNC5B receptor deletion exacerbates tissue injury in response to AKI.

UNC5B receptor deletion exacerbates tissue injury in response to AKI.

http://www.wikidata.org/entity/Q37524105

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Netrin-1 as a novel therapeutic target in cardiovascular disease: to activate or inhibit?Guidance cue netrin-1 and the regulation of inflammation in acute and chronic kidney disease Cellular and Molecular Mechanisms of AKI.UNC5B receptor deletion exacerbates DSS-induced colitis in mice by increasing epithelial cell apoptosis.Deletion of UNC5B in Kidney Epithelium Exacerbates Diabetic Nephropathy in Mice MicroRNA-150 deletion in mice protects kidney from myocardial infarction-induced acute kidney injury.Honey feeding protects kidney against cisplatin nephrotoxicity through suppression of inflammation.Netrin as a Novel Biomarker and Its Therapeutic Implications in Diabetes Mellitus and Diabetes-Associated Complications

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UNC5B receptor deletion exacerbates tissue injury in response to AKI.

http://www.wikidata.org/entity/Q37524105

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 10 October 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

UNC5B receptor deletion exacerbates tissue injury in response to AKI.

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UNC5B receptor deletion exacerbates tissue injury in response to AKI.

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type

label

UNC5B receptor deletion exacerbates tissue injury in response to AKI.

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UNC5B receptor deletion exacerbates tissue injury in response to AKI.

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prefLabel

UNC5B receptor deletion exacerbates tissue injury in response to AKI.

@en

UNC5B receptor deletion exacerbates tissue injury in response to AKI.

@nl

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Journal of the American Society of Nephrology

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UNC5B receptor deletion exacerbates tissue injury in response to AKI.

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Calpurnia Jayakumar

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Dean Y Li

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Ganesan Ramesh

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Punithavathi Ranganathan

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Sutip Navankasattusas

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Acute renal failure - definition, outcome measures, animal models, fluid therapy and information technology needs: the Second International Consensus Conference of the Acute Dialysis Quality Initiative (ADQI) Group

Netrin-1 mediates neuronal survival through PIKE-L interaction with the dependence receptor UNC5B

Netrin-1 inhibits leukocyte migration in vitro and in vivo.

The dependence receptor UNC5H2 mediates apoptosis through DAP-kinase

Netrin-1 prevents ischemia/reperfusion-induced myocardial infarction via a DCC/ERK1/2/eNOS s1177/NO/DCC feed-forward mechanism

p53RDL1 regulates p53-dependent apoptosis

The dependence receptor hypothesis

Netrin-1 and its receptors in tumorigenesis

Evidence that fibroblasts derive from epithelium during tissue fibrosis

The molecular biology of axon guidance

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239-249

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scholarly article

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10.1681/ASN.2013040418

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2

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25

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2013-10-10T00:00:00Z

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24115477

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3904565

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