Aging is not programmed: genetic pseudo-program is a shadow of developmental growth. - Wikidata - awgldk - TriplyDB

Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

http://www.wikidata.org/entity/Q37525814

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Principles of alternative gerontology Reprogramming of energy metabolism as a driver of aging Long live FOXO: unraveling the role of FOXO proteins in aging and longevity Milk--A Nutrient System of Mammalian Evolution Promoting mTORC1-Dependent Translation The many ways to age for a single yeast cell Computer-aided discovery of biological activity spectra for anti-aging and anti-cancer olive oil oleuropeins.A meta-analysis on age-associated changes in blood DNA methylation: results from an original analysis pipeline for Infinium 450k data.Epigenetic stress responses induce muscle stem-cell ageing by Hoxa9 developmental signals.From rapalogs to anti-aging formula.Wound healing and longevity: lessons from long-lived αMUPA mice Ageing-associated changes in the human DNA methylome: genomic locations and effects on gene expression.Why is aging conserved and what can we do about it?Energy excess is the main cause of accelerated aging of mammals.Synchronized age-related gene expression changes across multiple tissues in human and the link to complex diseases.Rejuvenating immunity: "anti-aging drug today" eight years later.Koschei the immortal and anti-aging drugs Length of paternal lifespan is manifested in the DNA methylome of their nonagenarian progeny.Identification of novel plasma glycosylation-associated markers of aging.Inverse correlation between longevity and developmental rate among wild C. elegans strains.Gerosuppression by pan-mTOR inhibitors.Enhancing the delivery of resveratrol in humans: if low bioavailability is the problem, what is the solution?Quasi-programmed aging of budding yeast: a trade-off between programmed processes of cell proliferation, differentiation, stress response, survival and death defines yeast lifespan.Tumour suppressors and cellular senescence.Gerometabolites: the pseudohypoxic aging side of cancer oncometabolites.A synopsis on aging-Theories, mechanisms and future prospects.Circulating miRNAs and miRNA shuttles as biomarkers: Perspective trajectories of healthy and unhealthy aging Keeping up with the Red Queen: the pace of aging as an adaptation.Age-associated vulval integrity is an important marker of nematode healthspan.Phase II neoadjuvant clinical trial of carboplatin and eribulin in women with triple negative early-stage breast cancer (NCT01372579).Adaptive sequence convergence of the tumor suppressor ADAMTS9 between small-bodied mammals displaying exceptional longevity.Somatic growth, aging, and longevity.FOXO Transcriptional Factors and Long-Term Living.The Continuum of Aging and Age-Related Diseases: Common Mechanisms but Different Rates.

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P2860

Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

http://www.wikidata.org/entity/Q37525814

description

2013 nî lūn-bûn

@nan

2013年の論文

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2013年学术文章

@wuu

2013年学术文章

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2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

@zh

2013年學術文章

@zh-hant

name

Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

@en

Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

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type

label

Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

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Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

@nl

prefLabel

Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

@en

Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

@nl

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Aging is not programmed: genetic pseudo-program is a shadow of developmental growth.

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P2860

Increased life span due to calorie restriction in respiratory-deficient yeast

Extension of chronological life span in yeast by decreased TOR pathway signaling

Disruption of the mouse mTOR gene leads to early postimplantation lethality and prohibits embryonic stem cell development

mTOR is essential for growth and proliferation in early mouse embryos and embryonic stem cells

Rapamycin, but not resveratrol or simvastatin, extends life span of genetically heterogeneous mice

S6K1(-/-)/S6K2(-/-) mice exhibit perinatal lethality and rapamycin-sensitive 5'-terminal oligopyrimidine mRNA translation and reveal a mitogen-activated protein kinase-dependent S6 kinase pathway

Lessons on longevity from budding yeast

mTOR is a key modulator of ageing and age-related disease

Aging as a particular case of phenoptosis, the programmed death of an organism (a response to Kirkwood and Melov "On the programmed/non-programmed nature of ageing within the life history")

Reduced TOR signaling extends chronological life span via increased respiration and upregulation of mitochondrial gene expression

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3736-3742

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scholarly article

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10.4161/CC.27188

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24

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12

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Mikhail Blagosklonny

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2013-11-15T00:00:00Z

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