MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2. - Wikidata - awgldk - TriplyDB

MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

http://www.wikidata.org/entity/Q37539979

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Mechanisms of nanosized titanium dioxide-induced testicular oxidative stress and apoptosis in male mice Protective role of murine β-defensins 3 and 4 and cathelin-related antimicrobial peptide in Fusarium solani keratitis Aspergillus and Fusarium corneal infections are regulated by Th17 cells and IL-17-producing neutrophils.Role of Pattern Recognition Receptors in the Modulation of Antimicrobial Peptide Expression in the Corneal Epithelial Innate Response to F. solani.Distinct roles for Dectin-1 and TLR4 in the pathogenesis of Aspergillus fumigatus keratitis Toll-like receptors involved in the pathogenesis of experimental Candida albicans keratitis A murine model of contact lens-associated fusarium keratitis.Dendritic cell physiology and function in the eye.Overview of vertebrate animal models of fungal infection.Characterization of fusarium keratitis outbreak isolates: contribution of biofilms to antimicrobial resistance and pathogenesis.Zinc and Manganese Chelation by Neutrophil S100A8/A9 (Calprotectin) Limits Extracellular Aspergillus fumigatus Hyphal Growth and Corneal Infection.IL-17A-mediated protection against Acanthamoeba keratitis.Topical flagellin-mediated innate defense against Candida albicans keratitis.Therapeutic administration of the chemokine CXCL1/KC abrogates autoimmune inflammatory heart disease Expression of innate and adaptive immune mediators in human corneal tissue infected with Aspergillus or fusarium.Indoleamine 2,3-Dioxygenase Is Involved in the Inflammation Response of Corneal Epithelial Cells to Aspergillus fumigatus Infections Resistance to HSV-1 infection in the epithelium resides with the novel innate sensor, IFI-16 Fungal antioxidant pathways promote survival against neutrophils during infection.Pattern recognition receptors in microbial keratitis JAK/STAT regulation of Aspergillus fumigatus corneal infections and IL-6/23-stimulated neutrophil, IL-17, elastase, and MMP9 activity.Expression and potential role of major inflammatory cytokines in experimental keratomycosis.Toll-like receptors at the ocular surface Host-pathogen interactions in the cornea.Toll-like receptors in ocular surface diseases: overview and new findings.The role of cytokines and pathogen recognition molecules in fungal keratitis - Insights from human disease and animal models.Current Thoughts in Fungal Keratitis: Diagnosis and Treatment.The RodA hydrophobin on Aspergillus fumigatus spores masks dectin-1- and dectin-2-dependent responses and enhances fungal survival in vivo.Pretreatment with lipopolysaccharide modulates innate immunity in corneal fibroblasts challenged with Aspergillus fumigatus.Role of MyD88 in adenovirus keratitis.Innate Immune Sensing of Fusarium culmorum by Mouse Dendritic Cells.Flagellin-induced expression of CXCL10 mediates direct fungal killing and recruitment of NK cells to the cornea in response to Candida albicans infection.Molecular mechanism of oxidative damage of lung in mice following exposure to lanthanum chloride.Pulmonary toxicity in mice following exposure to cerium chloride.Application of image recognition-based automatic hyphae detection in fungal keratitis.Inflammation and oxidative stress in corneal tissue in experimental keratitis due to Fusarium solani: Amelioration following topical therapy with voriconazole and epigallocatechin gallate.IL-17 plays a central role in initiating experimental Candida albicans infection in mouse corneas.Toll-like receptor-associated keratitis and strategies for its management.Atovaquone Impairs Growth of Aspergillus and Fusarium Keratitis Isolates by Modulating Mitochondrial Function and Zinc Homeostasis.Protein Deiminase 4 and CR3 Regulate Aspergillus fumigatus and β-Glucan-Induced Neutrophil Extracellular Trap Formation, but Hyphal Killing Is Dependent Only on CR3.Neutrophil Caspase-11 Is Required for Cleavage of Caspase-1 and Secretion of IL-1β in Infection

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MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

http://www.wikidata.org/entity/Q37539979

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on July 2008

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

@en

MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

@nl

type

label

MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

@en

MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

@nl

prefLabel

MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

@en

MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

@nl

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JIMMUNOL.181.1.593

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Journal of Immunology

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MyD88 regulation of Fusarium keratitis is dependent on TLR4 and IL-1R1 but not TLR2.

@en

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Ahmad B Tarabishy

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Bishr Aldabagh

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Eric Pearlman

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Jonathan H Lass

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Mahmoud A Ghannoum

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Pranab K Mukherjee

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Yan Sun

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Yoshifumi Imamura

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P2860

Fusarium and Candida albicans biofilms on soft contact lenses: model development, influence of lens type, and susceptibility to lens care solutions

The human beta-glucan receptor is widely expressed and functionally equivalent to murine Dectin-1 on primary cells

Severe impairment in early host defense against Candida albicans in mice deficient in myeloperoxidase

Posaconazole as Salvage Treatment for Invasive Fusariosis in Patients with Underlying Hematologic Malignancy and Other Conditions

Innate immunity to the pathogenic fungus Coccidioides posadasii is dependent on Toll-like receptor 2 and Dectin-1.

The corneal stroma is endowed with a significant number of resident dendritic cells.

Immune sensing of Candida albicans requires cooperative recognition of mannans and glucans by lectin and Toll-like receptors

Members of the Fusarium solani species complex that cause infections in both humans and plants are common in the environment.

An integrated model of the recognition of Candida albicans by the innate immune system.

Pathogenic mechanisms of P. aeruginosa keratitis: a review of the role of T cells, Langerhans cells, PMN, and cytokines.

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593-600

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10.4049/JIMMUNOL.181.1.593

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1

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181

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2008-07-01T00:00:00Z

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5289015

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18566426

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Toll-like receptor

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3909484

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