TGF-beta signaling in chondrocyte terminal differentiation and osteoarthritis: modulation and integration of signaling pathways through receptor-Smads.
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The Regulatory Role of Signaling Crosstalk in Hypertrophy of MSCs and Human Articular ChondrocytesThe chondrocyte clock gene Bmal1 controls cartilage homeostasis and integrity.Insights on Molecular Mechanisms of Chondrocytes Death in OsteoarthritisInhibition of TGF-β signaling in mesenchymal stem cells of subchondral bone attenuates osteoarthritisInference of dynamical gene-regulatory networks based on time-resolved multi-stimuli multi-experiment data applying NetGenerator V2.0.Transplantation of human Wharton's jelly-derived mesenchymal stem cells highly expressing TGFβ receptors in a rabbit model of disc degeneration.Cellular senescence in aging and osteoarthritis.Integrin α1β1 differentially regulates cytokine-mediated responses in chondrocytes.F-spondin deficient mice have a high bone mass phenotype.Targeting TGFβ signaling in subchondral bone and articular cartilage homeostasisECM stiffness primes the TGFβ pathway to promote chondrocyte differentiation.Growth factor regulation of proliferation and survival of multipotential stromal cells.Characterization of microRNA expression profiles in normal and osteoarthritic human chondrocytes.Circadian Clocks in Articular Cartilage and Bone: A Compass in the Sea of Matrices.Mouse limb skeletal growth and synovial joint development are coordinately enhanced by Kartogenin.TGF-β signaling and the development of osteoarthritisDynamic modelling of microRNA regulation during mesenchymal stem cell differentiation.Characterization of the cartilage DNA methylome in knee and hip osteoarthritisCurrent surgical treatment of knee osteoarthritisStrategies to minimize hypertrophy in cartilage engineering and regeneration.A Semiquantitative Framework for Gene Regulatory Networks: Increasing the Time and Quantitative Resolution of Boolean Networks.Gene-gene interactions between TGF-β/Smad3 signalling pathway polymorphisms affect susceptibility to knee osteoarthritis.Kindlin-2 controls TGF-β signalling and Sox9 expression to regulate chondrogenesisNeuroleukin/Autocrine Motility Factor Receptor Pathway Promotes Proliferation of Articular Chondrocytes through Activation of AKT and Smad2/3.Smad2 and Smad3 Regulate Chondrocyte Proliferation and Differentiation in the Growth Plate.Fuyuan Decoction Enhances SOX9 and COL2A1 Expression and Smad2/3 Phosphorylation in IL-1β-Activated ChondrocytesHedgehog signalling does not stimulate cartilage catabolism and is inhibited by Interleukin-1βAnti-RANKL treatment inhibits erosive joint destruction and lowers inflammation but has no effect on bone formation in the delayed-type hypersensitivity arthritis (DTHA) modelCircular RNA Related to the Chondrocyte ECM Regulates MMP13 Expression by Functioning as a MiR-136 'Sponge' in Human Cartilage Degradation.Cathepsin-Mediated Alterations in TGFß-Related Signaling Underlie Disrupted Cartilage and Bone Maturation Associated With Impaired Lysosomal Targeting.Harnessing cell–biomaterial interactions for osteochondral tissue regeneration.Role of Chondrocytes in Cartilage Formation, Progression of Osteoarthritis and Cartilage Regeneration.Calcitonin attenuates cartilage degeneration and nociception in an experimental rat model of osteoarthritis: role of TGF-β in chondrocytesBasic science of osteoarthritis.Expression of TGF-β Signaling Regulator RBPMS (RNA-Binding Protein With Multiple Splicing) Is Regulated by IL-1β and TGF-β Superfamily Members, and Decreased in Aged and Osteoarthritic Cartilage.Deletion of the transforming growth factor β receptor type II gene in articular chondrocytes leads to a progressive osteoarthritis-like phenotype in mice.Nanosecond pulsed electric fields (nsPEFs) regulate phenotypes of chondrocytes through Wnt/β-catenin signaling pathway.TGFβ signaling in cartilage development and maintenance.Synovial TGF-β1 and MMP-3 levels and their correlation with the progression of temporomandibular joint osteoarthritis combined with disc displacement: A preliminary study.Association between Single Nucleotide Polymorphisms of SMAD3 and BMP5 with the Risk of Knee Osteoarthritis.
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P2860
TGF-beta signaling in chondrocyte terminal differentiation and osteoarthritis: modulation and integration of signaling pathways through receptor-Smads.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 26 June 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
TGF-beta signaling in chondroc ...... thways through receptor-Smads.
@en
TGF-beta signaling in chondroc ...... thways through receptor-Smads.
@nl
type
label
TGF-beta signaling in chondroc ...... thways through receptor-Smads.
@en
TGF-beta signaling in chondroc ...... thways through receptor-Smads.
@nl
prefLabel
TGF-beta signaling in chondroc ...... thways through receptor-Smads.
@en
TGF-beta signaling in chondroc ...... thways through receptor-Smads.
@nl
P2093
P1476
TGF-beta signaling in chondroc ...... thways through receptor-Smads.
@en
P2093
E N Blaney Davidson
P M van der Kraan
W B van den Berg
P304
P356
10.1016/J.JOCA.2009.06.008
P577
2009-06-26T00:00:00Z