Inositol trisphosphate modification of ion transport in rough endoplasmic reticulum.
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Cloning of human Ca2+ homoeostasis endoplasmic reticulum protein (CHERP): regulated expression of antisense cDNA depletes CHERP, inhibits intracellular Ca2+ mobilization and decreases cell proliferationAntisense-mediated loss of calcium homoeostasis endoplasmic reticulum protein (CHERP; ERPROT213-21) impairs Ca2+ mobilization, nuclear factor of activated T-cells (NFAT) activation and cell proliferation in Jurkat T-lymphocytesTherapeutic potential for novel drugs targeting the type 1 cholecystokinin receptorCalcium entry into the inositol 1,4,5-trisphosphate-releasable calcium pool is mediated by a GTP-regulatory mechanism.Recent advances in the treatment of duodenal ulcer disease. A surgical perspective.Physiological localization of an agonist-sensitive pool of Ca2+ in parotid acinar cells.Volume-sensitive K-Cl cotransport in inside-out vesicles made from erythrocyte membranes from sheep of low-K phenotype.Effects of caerulein on the apical cytoskeleton of the pancreatic acinar cell.Stimulation of inositol trisphosphate and diacylglycerol production in renal tubular cells by parathyroid hormone.Signal transduction mechanisms involved in hormonal Ca2+ fluxes.Calbindin-D9k Ablation Disrupt Glucose/Pancreatic Insulin Homeostasis'Quantal' Ca(2+) release reassessed--a clue to oscillation and synchronization.Second messenger systems and adaptation.Localization of calcium and microfilament changes in mechanically stressed cells.Second messengers and the regulation of Ca2+ fluxes by Ca2+-mobilizing agonists in rat liver.Inositol trisphosphate as a second messenger in signal transduction.Ca2+ release and Ca2+ entry induced by rapid cytosolic alkalinization in Jurkat T-lymphocytes.Regulation of agonist-evoked [Ca2+]i oscillation by intracellular Ca2+ and Ba2+ in AR42J cells.Intracellular calcium pools in neuroblastoma x glioma hybrid NG108-15 cells.Estimation of the free [Ca2+] gradient across endoplasmic reticulum membranes by a null-point method.Liver inositol, 1,4,5-trisphosphate-binding sites are the Ca2(+)-mobilizing receptorsPhysiological concentrations of inorganic phosphate affect MgATP-dependent Ca2+ storage and inositol trisphosphate-induced Ca2+ efflux in microsomal vesicles from non-hepatic cells.MgATP-dependent accumulation of calcium ions and inorganic phosphate in a liver reticular pool.Ca2+ release by inositol 1,4,5-trisphosphate is blocked by the K(+)-channel blockers apamin and tetrapentylammonium ion, and a monoclonal antibody to a 63 kDa membrane protein: reversal of blockade by K+ ionophores nigericin and valinomycin and puriThe mechanism of action of GTP on Ca2+ efflux from rat liver microsomal vesicles.Potassium-channel blockers inhibit inositol trisphosphate-induced calcium release in the microsomal fractions isolated from the rat brain.Analysis of a novel double-barreled anion channel from rat liver rough endoplasmic reticulum.Molecular target sizes of inositol 1,4,5-trisphosphate receptors in liver and cerebellum.Differences between the effects of cromakalim and nifedipine on agonist-induced responses in rabbit aorta.Characteristics of the contractile response of rabbit aorta produced by cromakalim in calcium-free solution.The size of inositol 1,4,5-trisphosphate-sensitive Ca2+ stores depends on inositol 1,4,5-trisphosphate concentrationRegulation of the cerebellar inositol 1,4,5-trisphosphate receptor by univalent cations.Luminal Ca2+ increases the affinity of inositol 1,4,5-trisphosphate for its receptor.Evidence that ATP-dependent Ca2+ transport in rat parotid microsomal membranes requires charge compensation.Two components of hormone-evoked calcium release from intracellular stores of pancreatic acinar cellsCharacterization of inositol 1,4,5-trisphosphate-stimulated calcium release from rat cerebellar microsomal fractions. Comparison with [3H]inositol 1,4,5-trisphosphate binding.Binding of inositol trisphosphate by a liver microsomal fraction.Effects of rubidium on responses to potassium channel openers in rat isolated aorta.Calcium-activated membrane depolarization via modulation of chloride efflux from parietal cells during gastrin stimulation.Calcium metabolism and bombesin-stimulated pancreatic enzyme secretion.
P2860
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P2860
Inositol trisphosphate modification of ion transport in rough endoplasmic reticulum.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on July 1985
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Inositol trisphosphate modification of ion transport in rough endoplasmic reticulum.
@en
Inositol trisphosphate modification of ion transport in rough endoplasmic reticulum.
@nl
type
label
Inositol trisphosphate modification of ion transport in rough endoplasmic reticulum.
@en
Inositol trisphosphate modification of ion transport in rough endoplasmic reticulum.
@nl
prefLabel
Inositol trisphosphate modification of ion transport in rough endoplasmic reticulum.
@en
Inositol trisphosphate modification of ion transport in rough endoplasmic reticulum.
@nl
P2093
P2860
P356
P1476
Inositol trisphosphate modification of ion transport in rough endoplasmic reticulum.
@en
P2093
P2860
P304
P356
10.1073/PNAS.82.13.4433
P407
P577
1985-07-01T00:00:00Z