MyD88 signaling is directly involved in the development of murine placental malaria. - Wikidata - awgldk - TriplyDB

MyD88 signaling is directly involved in the development of murine placental malaria.

MyD88 signaling is directly involved in the development of murine placental malaria.

http://www.wikidata.org/entity/Q37546441

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Interferons and interferon regulatory factors in malaria Differential roles of inflammation and apoptosis in initiation of mid-gestational abortion in malaria-infected C57BL/6 and A/J mice.TLR4-Mediated Placental Pathology and Pregnancy Outcome in Experimental Malaria.IFNGR1 signaling is associated with adverse pregnancy outcomes during infection with malaria parasites.

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MyD88 signaling is directly involved in the development of murine placental malaria.

http://www.wikidata.org/entity/Q37546441

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 09 December 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

MyD88 signaling is directly involved in the development of murine placental malaria.

@en

MyD88 signaling is directly involved in the development of murine placental malaria.

@nl

type

label

MyD88 signaling is directly involved in the development of murine placental malaria.

@en

MyD88 signaling is directly involved in the development of murine placental malaria.

@nl

prefLabel

MyD88 signaling is directly involved in the development of murine placental malaria.

@en

MyD88 signaling is directly involved in the development of murine placental malaria.

@nl

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Infection and Immunity

P1476

MyD88 signaling is directly involved in the development of murine placental malaria.

@en

P2093

José Maria Alvarez

http://www.w3.org/2001/XMLSchema#string

Keitty Raquel Benevides Pereira

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Leandro Gustavo da Silva

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Lutero Hasenkamp

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Maria Regina D'Império Lima

http://www.w3.org/2001/XMLSchema#string

Renato Barboza

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: cell signaling receptors, glycosylphosphatidylinositol (GPI) structural requirement, and regulation of GPI activity

Toll-like receptors and their crosstalk with other innate receptors in infection and immunity

High efficiency transfection of Plasmodium berghei facilitates novel selection procedures

Distinct placental malaria pathology caused by different Plasmodium berghei lines that fail to induce cerebral malaria in the C57BL/6 mouse.

Pregnancy outcome and placenta pathology in Plasmodium berghei ANKA infected mice reproduce the pathogenesis of severe malaria in pregnant women.

The molecular pathophysiology of bacterially induced preterm labor: insights from the murine model.

Recrudescent Plasmodium berghei from pregnant mice displays enhanced binding to the placenta and induces protection in multigravida

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P304

830-838

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scholarly article

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10.1128/IAI.01288-13

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2

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82

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Silvia B Boscardin

Claudio Romero Farias Marinho

Aramys Silva Reis

Sabrina Epiphanio

Fabio T M Costa

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2013-12-09T00:00:00Z

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24478096

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maternal health

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3911391

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