Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
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Modulation of inflammasome activity by Porphyromonas gingivalis in periodontitis and associated systemic diseasesPeriodontal Disease-Induced Atherosclerosis and Oxidative StressThe Biology of Chronic Graft-versus-Host Disease: A Task Force Report from the National Institutes of Health Consensus Development Project on Criteria for Clinical Trials in Chronic Graft-versus-Host DiseaseBacterial recognition pathways that lead to inflammasome activationLessons from Microglia Aging for the Link between Inflammatory Bone Disorders and Alzheimer's DiseaseThe DNA Sensor AIM2 Maintains Intestinal Homeostasis via Regulation of Epithelial Antimicrobial Host Defense.Porphyromonas gingivalis fimbriae dampen P2X7-dependent interleukin-1β secretion.DNA-sensing inflammasomes: regulation of bacterial host defense and the gut microbiota.Dental Calculus Stimulates Interleukin-1β Secretion by Activating NLRP3 Inflammasome in Human and Mouse Phagocytes.Cholesterol crystals enhance TLR2- and TLR4-mediated pro-inflammatory cytokine responses of monocytes to the proatherogenic oral bacterium Porphyromonas gingivalisEpstein-Barr virus infection-induced inflammasome activation in human monocytes.Microbial Nucleic Acid Sensing in Oral and Systemic Diseases.AIM2 inflammasome in infection, cancer, and autoimmunity: Role in DNA sensing, inflammation, and innate immunityPorphyromonas gingivalis-induced miR-132 regulates TNFα expression in THP-1 derived macrophages.Heterogeneous Porphyromonas gingivalis LPS modulates immuno-inflammatory response, antioxidant defense and cytoskeletal dynamics in human gingival fibroblasts.Sterol Regulatory Element-Binding Protein-1c Regulates Inflammasome Activation in Gingival Fibroblasts Infected with High-Glucose-Treated Porphyromonas gingivalis.Collaborative action of Toll-like and NOD-like receptors as modulators of the inflammatory response to pathogenic bacteria.Danger- and pathogen-associated molecular patterns recognition by pattern-recognition receptors and ion channels of the transient receptor potential family triggers the inflammasome activation in immune cells and sensory neurons.The inflammasome and danger molecule signaling: at the crossroads of inflammation and pathogen persistence in the oral cavity.Metabolic Remodeling, Inflammasome Activation, and Pyroptosis in Macrophages Stimulated by Porphyromonas gingivalis and Its Outer Membrane Vesicles.Involvement of NLRP10 in IL-1α induction of oral epithelial cells by periodontal pathogens.NLR-regulated pathways in cancer: opportunities and obstacles for therapeutic interventions.The role of Toll-like receptors in periodontitis.Common Polymorphisms in IFI16 and AIM2 Genes Are Associated With Periodontal Disease.CD24 activates the NLRP3 inflammasome through c-Src kinase activity in a model of the lining epithelium of inflamed periodontal tissues.AIM2 contributes to the maintenance of intestinal integrity via Akt and protects against Salmonella mucosal infection.Porphyromonas gingivalis induces autophagy in THP-1-derived macrophages.Genome-wide transcriptome induced by Porphyromonas gingivalis LPS supports the notion of host-derived periodontal destruction and its association with systemic diseases.NLRP3 inflammasome mediates interleukin-1β production in immune cells in response to Acinetobacter baumannii and contributes to pulmonary inflammation in mice.Regulation of the NLRP3 inflammasome in Porphyromonas gingivalis-accelerated periodontal disease.Porphyromonas gingivalis Differentially Modulates Cell Death Profile in Ox-LDL and TNF-α Pre-Treated Endothelial Cells.Aggregatibacter actinomycetemcomitans-Induced AIM2 Inflammasome Activation Is Suppressed by Xylitol in Differentiated THP-1 Macrophages.Porphyromonas gingivalis and Epstein-Barr Virus Are Associated With Increased Levels of Visfatin in Gingival Crevicular Fluid.Contradictory roles of Porphyromonas gingivalis gingipains in caspase-1 activation.Activation of the NLRP3 inflammasome in Porphyromonas gingivalis-accelerated atherosclerosis.Outer Membrane Vesicles Prime and Activate Macrophage Inflammasomes and Cytokine Secretion In Vitro and In Vivo.MicroRNA-132 Negatively Regulates Palmitate-Induced NLRP3 Inflammasome Activation through FOXO3 Down-Regulation in THP-1 Cells.New evidence of connections between increased O-GlcNAcylation and inflammasome in the oral mucosa of patients with oral lichen planus.Caspase-4 activation by a bacterial surface protein is mediated by cathepsin G in human gingival fibroblasts.The P2X7 Receptor in Inflammatory Diseases: Angel or Demon?
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P2860
Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 14 October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
@en
Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
@nl
type
label
Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
@en
Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
@nl
prefLabel
Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
@en
Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
@nl
P2093
P2860
P356
P1476
Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection.
@en
P2093
Eunjoo Park
Hee Sam Na
Seong Yeol Shin
Yu-Ri Song
P2860
P304
P356
10.1128/IAI.00862-13
P407
P50
P577
2013-10-14T00:00:00Z