The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction.
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How Biomaterials Can Influence Various Cell Types in the Repair and Regeneration of the Heart after Myocardial InfarctionThe circular relationship between matrix metalloproteinase-9 and inflammation following myocardial infarctionUsing proteomics to uncover extracellular matrix interactions during cardiac remodeling.Physiological Implications of Myocardial Scar StructureDirect isolation of myofibroblasts and fibroblasts from bleomycin-injured lungs reveals their functional similarities and differencesInterleukin-1 beta influences on lysyl oxidases and matrix metalloproteinases profile of injured anterior cruciate ligament and medial collateral ligament fibroblastsProteins involved in platelet signaling are differentially regulated in acute coronary syndrome: a proteomic studyEpigenetic Regulation of Matrix Metalloproteinase-1 and -3 Expression in Mycobacterium tuberculosis Infection.Molecular Imaging of Healing After Myocardial Infarction.Cardiac fibroblast: the renaissance cell.Cell adhesion molecule mediation of myocardial inflammatory responses associated with ventricular pacing.Human embryonic stem cell-derived cardiomyocytes migrate in response to gradients of fibronectin and Wnt5aReversible and irreversible differentiation of cardiac fibroblasts.High molecular weight fibroblast growth factor-2 in the human heart is a potential target for prevention of cardiac remodelingCellular mechanisms of tissue fibrosis. 6. Purinergic signaling and response in fibroblasts and tissue fibrosisMembrane-associated matrix proteolysis and heart failure.Experimental and computational investigation of altered mechanical properties in myocardium after hydrogel injection.Systemic approach to identify serum microRNAs as potential biomarkers for acute myocardial infarction.Matricellular proteins and matrix metalloproteinases mark the inflammatory and fibrotic response in human cardiac allograft rejection.The zebrafish heart regenerates after cryoinjury-induced myocardial infarction.Emerging roles of fibronectin in thrombosis.Expression and function of osteopontin in vascular adventitial fibroblasts and pathological vascular remodelingThe role of inflammatory and fibrogenic pathways in heart failure associated with aging.Effect of intracoronary delivery of autologous bone marrow mononuclear cells 2 to 3 weeks following acute myocardial infarction on left ventricular function: the LateTIME randomized trial.Myocardial tissue elastic properties determined by atomic force microscopy after stromal cell-derived factor 1α angiogenic therapy for acute myocardial infarction in a murine model.A proteome comparison between physiological angiogenesis and angiogenesis in glioblastoma.Inhibition of Let-7 microRNA attenuates myocardial remodeling and improves cardiac function postinfarction in mice.Spatial anisotropies and temporal fluctuations in extracellular matrix network texture during early embryogenesis.Periostin: a matricellular protein involved in peritoneal injury during peritoneal dialysis.Transcriptional profiling of left ventricle and peripheral blood mononuclear cells in a rat model of postinfarction heart failure.Sodium hydrosulfide prevents myocardial dysfunction through modulation of extracellular matrix accumulation and vascular densityStretching fibronectin fibres disrupts binding of bacterial adhesins by physically destroying an epitopeCardiac mesenchymal stem cells contribute to scar formation after myocardial infarction.Differential roles of cardiac and leukocyte derived macrophage migration inhibitory factor in inflammatory responses and cardiac remodelling post myocardial infarction.Dynamic molecular and histopathological changes in the extracellular matrix and inflammation in the transition to heart failure in isolated volume overloadApoptosis-like cell death induction and aberrant fibroblast properties in human incisional hernia fascia.Obesity superimposed on aging magnifies inflammation and delays the resolving response after myocardial infarctionEffect and mechanism of thrombospondin-1 on the angiogenesis potential in human endothelial progenitor cells: an in vitro studyAssociation of serum periostin with cardiac function and short-term prognosis in acute myocardial infarction patientsPolymorphisms of the LTA gene may contribute to the risk of myocardial infarction: a meta-analysis.
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The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 23 July 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
The extracellular matrix as a ...... llowing myocardial infarction.
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The extracellular matrix as a ...... llowing myocardial infarction.
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type
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The extracellular matrix as a ...... llowing myocardial infarction.
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The extracellular matrix as a ...... llowing myocardial infarction.
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prefLabel
The extracellular matrix as a ...... llowing myocardial infarction.
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The extracellular matrix as a ...... llowing myocardial infarction.
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P2093
P2860
P921
P1476
The extracellular matrix as a ...... llowing myocardial infarction.
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P2093
Carlos Gonzalez-Quesada
Marcin Dobaczewski
Nikolaos G Frangogiannis
P2860
P304
P356
10.1016/J.YJMCC.2009.07.015
P577
2009-07-23T00:00:00Z