In vitro evidence that covalent crosslinking of neurofilaments occurs in gamma-diketone neuropathy.
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Mechanisms of toxic injury in the peripheral nervous system: neuropathologic considerationsTargeted disruption of the cyclin-dependent kinase 5 gene results in abnormal corticogenesis, neuronal pathology and perinatal deathFamilial amyotrophic lateral sclerosis with a point mutation of SOD-1: intrafamilial heterogeneity of disease duration associated with neurofibrillary tangles.Disruption of the NF-H gene increases axonal microtubule content and velocity of neurofilament transport: relief of axonopathy resulting from the toxin beta,beta'-iminodipropionitrile.Pathogenetic studies of hexane and carbon disulfide neurotoxicity.Acrylamide arrests mitosis and prevents chromosome migration in the absence of changes in spindle microtubules.
P2860
In vitro evidence that covalent crosslinking of neurofilaments occurs in gamma-diketone neuropathy.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on August 1984
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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In vitro evidence that covalen ...... in gamma-diketone neuropathy.
@en
In vitro evidence that covalen ...... in gamma-diketone neuropathy.
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type
label
In vitro evidence that covalen ...... in gamma-diketone neuropathy.
@en
In vitro evidence that covalen ...... in gamma-diketone neuropathy.
@nl
prefLabel
In vitro evidence that covalen ...... in gamma-diketone neuropathy.
@en
In vitro evidence that covalen ...... in gamma-diketone neuropathy.
@nl
P2093
P2860
P356
P1476
In vitro evidence that covalen ...... in gamma-diketone neuropathy.
@en
P2093
D C Anthony
D G Graham
G Szakál-Quin
J W Priest
P2860
P304
P356
10.1073/PNAS.81.15.4979
P407
P577
1984-08-01T00:00:00Z