The ubiquitin-editing enzyme A20 (TNFAIP3) is a central regulator of immunopathology.
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Proof-of-concept, randomized, controlled clinical trial of Bacillus-Calmette-Guerin for treatment of long-term type 1 diabetesCharting the NF-κB pathway interactome mapContext-Dependent Regulation of Autophagy by IKK-NF-κB Signaling: Impact on the Aging ProcessA20 inhibits LUBAC-mediated NF-κB activation by binding linear polyubiquitin chains via its zinc finger 7Autophagy-Related Deubiquitinating Enzymes Involved in Health and DiseaseSelected Aspects in the Pathogenesis of Autoimmune DiseasesThe immunogenetics of primary biliary cirrhosis: A comprehensive reviewToll-like receptors in systemic lupus erythematosus: potential targets for therapeutic interventionDeconstructing innate immune signaling in myelodysplastic syndromesUbiquitin in the immune systemA20 (TNFAIP3) alleviates CVB3-induced myocarditis via inhibiting NF-κB signalingSpecific recognition of linear polyubiquitin by A20 zinc finger 7 is involved in NF-κB regulationOTUB1 overexpression in mesangial cells is a novel regulator in the pathogenesis of glomerulonephritis through the decrease of DCN levelNF-κB Pathway in Autoinflammatory Diseases: Dysregulation of Protein Modifications by Ubiquitin Defines a New Category of Autoinflammatory Diseases.Three single nucleotide polymorphisms of TNFAIP3 gene increase the risk of rheumatoid arthritis.rs10499194 polymorphism in the tumor necrosis factor-α inducible protein 3 (TNFAIP3) gene is associated with type-1 autoimmune hepatitis risk in Chinese Han population.Single nucleotide polymorphisms in TNFAIP3 were associated with the risks of rheumatoid arthritis in northern Chinese Han population.HSCARG downregulates NF-κB signaling by interacting with USP7 and inhibiting NEMO ubiquitination.TAX1BP1 and A20 inhibit antiviral signaling by targeting TBK1-IKKi kinasesZinc modulates the innate immune response in vivo to polymicrobial sepsis through regulation of NF-kappaBRecent advances in the genetics of systemic lupus erythematosus.Enterocyte-specific A20 deficiency sensitizes to tumor necrosis factor-induced toxicity and experimental colitis.Negative regulation of the NLRP3 inflammasome by A20 protects against arthritisTNFAIP3 maintains intestinal barrier function and supports epithelial cell tight junctions.A targeted association study in systemic lupus erythematosus identifies multiple susceptibility alleles.Interactive transcriptome analysis of malaria patients and infecting Plasmodium falciparum.A20 (Tnfaip3) deficiency in myeloid cells protects against influenza A virus infection.Profiling of immune-related microRNA expression in human cord blood and adult peripheral blood cells upon proinflammatory stimulation.A critical review of the molecular pathophysiology of lenalidomide sensitivity in 5q - myelodysplastic syndromes.Genetic association study of TNFAIP3, IFIH1, IRF5 polymorphisms with polymyositis/dermatomyositis in Chinese Han population.Deletion of the TNFAIP3/A20 gene detected by FICTION analysis in classical Hodgkin lymphomaCharacteristics of CARMA1-BCL10-MALT1-A20-NF-κB expression in T cell-acute lymphocytic leukemiaTumor suppressor A20 protects against cardiac hypertrophy and fibrosis by blocking transforming growth factor-beta-activated kinase 1-dependent signalingGenetic relationships between A20/TNFAIP3, chronic inflammation and autoimmune disease.Influence of vitamin D status and vitamin D3 supplementation on genome wide expression of white blood cells: a randomized double-blind clinical trial.Noise decomposition of intracellular biochemical signaling networks using nonequivalent reportersAnalysis of gender differences in genetic risk: association of TNFAIP3 polymorphism with male childhood-onset systemic lupus erythematosus in the Japanese populationProteomic analysis of differentially expressed proteins in vitamin C-treated AGS cells.A20 (TNFAIP3) genetic alterations in EBV-associated AIDS-related lymphoma.Commensal microbiota contributes to chronic endocarditis in TAX1BP1 deficient mice.
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P2860
The ubiquitin-editing enzyme A20 (TNFAIP3) is a central regulator of immunopathology.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 28 July 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
The ubiquitin-editing enzyme A20 (TNFAIP3) is a central regulator of immunopathology.
@en
The ubiquitin-editing enzyme A20
@nl
type
label
The ubiquitin-editing enzyme A20 (TNFAIP3) is a central regulator of immunopathology.
@en
The ubiquitin-editing enzyme A20
@nl
prefLabel
The ubiquitin-editing enzyme A20 (TNFAIP3) is a central regulator of immunopathology.
@en
The ubiquitin-editing enzyme A20
@nl
P356
P1433
P1476
The ubiquitin-editing enzyme A20 (TNFAIP3) is a central regulator of immunopathology.
@en
P2093
Geert van Loo
Lars Vereecke
P304
P356
10.1016/J.IT.2009.05.007
P50
P577
2009-07-28T00:00:00Z