Potentiation of Calcium Influx and Insulin Secretion in Pancreatic Beta Cell by the Specific TREK-1 Blocker Spadin. - Wikidata - awgldk - TriplyDB

Potentiation of Calcium Influx and Insulin Secretion in Pancreatic Beta Cell by the Specific TREK-1 Blocker Spadin.

Potentiation of Calcium Influx and Insulin Secretion in Pancreatic Beta Cell by the Specific TREK-1 Blocker Spadin.

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Identification and characterization of two zebrafish Twik related potassium channels, Kcnk2a and Kcnk2b

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Potentiation of Calcium Influx and Insulin Secretion in Pancreatic Beta Cell by the Specific TREK-1 Blocker Spadin.

http://www.wikidata.org/entity/Q37571762

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article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 25 December 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Potentiation of Calcium Influx ...... pecific TREK-1 Blocker Spadin.

@en

Potentiation of Calcium Influx ...... pecific TREK-1 Blocker Spadin.

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Potentiation of Calcium Influx ...... pecific TREK-1 Blocker Spadin.

@en

Potentiation of Calcium Influx ...... pecific TREK-1 Blocker Spadin.

@nl

prefLabel

Potentiation of Calcium Influx ...... pecific TREK-1 Blocker Spadin.

@en

Potentiation of Calcium Influx ...... pecific TREK-1 Blocker Spadin.

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Journal of Diabetes Research

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Potentiation of Calcium Influx ...... Specific TREK-1 Blocker Spadin

@en

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Alaeddine Djillani

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Catherine Heurteaux

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Christelle Devader

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Céline Hivelin

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Marc Borsotto

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Sophie Béraud-Dufour

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Sébastien Moreno

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Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patches

A mammalian two pore domain mechano-gated S-like K+ channel

Targeting cAMP/PKA pathway for glycemic control and type 2 diabetes therapy

Spadin, a sortilin-derived peptide, targeting rodent TREK-1 channels: a new concept in the antidepressant drug design.

Inhalational anesthetics activate two-pore-domain background K+ channels

Sequential phosphorylation mediates receptor- and kinase-induced inhibition of TREK-1 background potassium channels

Expression of the mechanosensitive 2PK+ channel TREK-1 in human osteoblasts

A TREK-1-like potassium channel in atrial cells inhibited by beta-adrenergic stimulation and activated by volatile anesthetics

Thermosensitivity of the two-pore domain K+ channels TREK-2 and TRAAK

Pancreatic β-cell-specific ablation of TASK-1 channels augments glucose-stimulated calcium entry and insulin secretion, improving glucose tolerance.

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3142175

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scholarly article

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10.1155/2016/3142175

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2016

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Thierry Coppola

Amar Abderrahmani

Hamid Moha Ou Maati

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2016-12-25T00:00:00Z

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28105440

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5220496

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