Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.
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Obesity, metabolic dysfunction, and cardiac fibrosis: pathophysiological pathways, molecular mechanisms, and therapeutic opportunitiesTherapeutic targeting of aldosterone: a novel approach to the treatment of glomerular diseaseAldosterone Induces Renal Fibrosis and Inflammatory M1-Macrophage Subtype via Mineralocorticoid Receptor in RatsGPER is involved in the stimulatory effects of aldosterone in breast cancer cells and breast tumor-derived endothelial cellsEmerging Concepts in the Molecular Basis of Pulmonary Arterial Hypertension: Part II: Neurohormonal Signaling Contributes to the Pulmonary Vascular and Right Ventricular Pathophenotype of Pulmonary Arterial HypertensionMineralocorticoid Receptor Antagonism for Cardiovascular Protection in End-Stage Renal Disease: New Data But the Controversy Continues.Natriuretic peptide receptor guanylyl cyclase-A pathway counteracts glomerular injury evoked by aldosterone through p38 mitogen-activated protein kinase inhibition.Salt Loading Promotes Kidney Injury via Fibrosis in Young Female Ren2 Rats.Racial differences in sensitivity of blood pressure to aldosteroneMineralocorticoid Receptor Signaling as a Therapeutic Target for Renal and Cardiac Fibrosis.Interfering with mineralocorticoid receptor activation: the past, present, and futureMedical therapy of stricturing Crohn's disease: what the gut can learn from other organs - a systematic review.Overnutrition, mTOR signaling, and cardiovascular diseases.Circulating aldosterone and natriuretic peptides in the general community: relationship to cardiorenal and metabolic disease.The pathophysiology of hypertension in patients with obesity.Indomethacin, amiloride, or eplerenone for treating hypokalemia in Gitelman syndromeMizoribine ameliorates renal injury and hypertension along with the attenuation of renal caspase-1 expression in aldosterone-salt-treated rats.TAK-242, a Toll-Like Receptor 4 Antagonist, Protects against Aldosterone-Induced Cardiac and Renal InjuryCeramide Production Mediates Aldosterone-Induced Human Umbilical Vein Endothelial Cell (HUVEC) Damages.Spironolactone treatment attenuates vascular dysfunction in type 2 diabetic mice by decreasing oxidative stress and restoring NO/GC signalingThe epidermal growth factor receptor and its ligands in cardiovascular disease.Elementary immunology: Na+ as a regulator of immunity.Renal denervation attenuates aldosterone expression and associated cardiovascular pathophysiology in angiotensin II-induced hypertension.Link between insulin resistance and hypertension: What is the evidence from evolutionary biology?Preclinical and Early Clinical Profile of a Highly Selective and Potent Oral Inhibitor of Aldosterone Synthase (CYP11B2).Pathophysiology and clinical presentations of salt-losing tubulopathies.Clinical Outcomes in Dialysis Patients: Prospects for Improvement with Aldosterone Receptor Antagonists.Uric acid promotes vascular stiffness, maladaptive inflammatory responses and proteinuria in western diet fed mice.Aldosterone and renin in cardiac patients referred for catheterization.NLRP3 inflammasome activation contributes to aldosterone-induced podocyte injury.Effects of mineralocorticoid receptor antagonists on left ventricular mass in chronic kidney disease patients: a systematic review and meta-analysis.Therapeutic compounds for Cushing's syndrome: a patent review (2012-2016).Radiofrequency ablation compared with laparoscopic adrenalectomy for aldosterone-producing adenoma.Caveolin 1-related autophagy initiated by aldosterone-induced oxidation promotes liver sinusoidal endothelial cells defenestration.A Novel Aldosterone Antagonist Limits Renal Injury in 5/6 Nephrectomy.Feedforward activation of endothelial ENaC by high sodium.Mineralocorticoid Receptor (MR) trans-Activation of Inflammatory AP-1 Signaling: DEPENDENCE ON DNA SEQUENCE, MR CONFORMATION, AND AP-1 FAMILY MEMBER EXPRESSIONThe roles of oxidative stress, endoplasmic reticulum stress, and autophagy in aldosterone/mineralocorticoid receptor-induced podocyte injury.Does Mineralocorticoid Receptor Antagonism Prevent Calcineurin Inhibitor-Induced Nephrotoxicity?Anti-inflammatory effects of Lefty-1 in renal tubulointerstitial inflammation via regulation of the NF-κB pathway.
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Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 18 June 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.
@en
Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.
@nl
type
label
Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.
@en
Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.
@nl
prefLabel
Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.
@en
Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.
@nl
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P1476
Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis.
@en
P2093
Nancy J Brown
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P2888
P304
P356
10.1038/NRNEPH.2013.110
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P577
2013-06-18T00:00:00Z