Breaking bad: manipulation of the host response by Porphyromonas gingivalis.
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Role of Porphyromonas gingivalis HmuY in Immunopathogenesis of Chronic PeriodontitisNeutrophil Functions in Periodontal HomeostasisModulation of inflammasome activity by Porphyromonas gingivalis in periodontitis and associated systemic diseasesHistorical and contemporary hypotheses on the development of oral diseases: are we there yet?Porphyromonas gingivalis and related bacteria: from colonial pigmentation to the type IX secretion system and gliding motilityPeriodontitis: from microbial immune subversion to systemic inflammationPorphyromonas gingivalis: An Overview of Periodontopathic Pathogen below the Gum LineNeutrophil homeostasis and inflammation: novel paradigms from studying periodontitisThe enduring importance of animal models in understanding periodontal diseaseGenetic and intervention studies implicating complement C3 as a major target for the treatment of periodontitis.Disruption of heterotypic community development by Porphyromonas gingivalis with small molecule inhibitors.Porphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis.Regulation of osteoclast homeostasis and inflammatory bone loss by MFG-E8.Neutrophil mobilization by surface-glycan altered Th17-skewing bacteria mitigates periodontal pathogen persistence and associated alveolar bone lossPorphyromonas gingivalis exacerbates ligature-induced, RANKL-dependent alveolar bone resorption via differential regulation of Toll-like receptor 2 (TLR2) and TLR4.The inflammophilic character of the periodontitis-associated microbiotaIn situ visualization of plasma cells producing antibodies reactive to Porphyromonas gingivalis in periodontitis: the application of the enzyme-labeled antigen method.Polymicrobial synergy and dysbiosis in inflammatory disease.Quantitative proteomics reveal distinct protein regulations caused by Aggregatibacter actinomycetemcomitans within subgingival biofilmsCigarette smoke condensate modulates migration of human gingival epithelial cells and their interactions with Porphyromonas gingivalis.The role of the microbiota in inflammation, carcinogenesis, and cancer therapy.Functional Advantages of Porphyromonas gingivalis Vesicles.Thinking globally, acting locally: harnessing the immune system to deal with recalcitrant pathogens.MFG-E8, a novel homeostatic regulator of osteoclastogenesis.Toll-Like Receptor 9-Mediated Inflammation Triggers Alveolar Bone Loss in Experimental Murine PeriodontitisPorphyromonas gingivalis within Placental Villous Mesenchyme and Umbilical Cord Stroma Is Associated with Adverse Pregnancy Outcome.Basic biology and role of interleukin-17 in immunity and inflammationPorphyromonas gingivalis virulence factors involved in subversion of leukocytes and microbial dysbiosisPG1058 Is a Novel Multidomain Protein Component of the Bacterial Type IX Secretion SystemFOXO responses to Porphyromonas gingivalis in epithelial cells.Porphyromonas gingivalis can invade periodontal ligament stem cells.Presence of Porphyromonas gingivalis in esophagus and its association with the clinicopathological characteristics and survival in patients with esophageal cancerThe expression of gingival epithelial junctions in response to subgingival biofilms.Human IDO-competent, long-lived immunoregulatory dendritic cells induced by intracellular pathogen, and their fate in humanized miceGenome Sequence of Porphyromonas gingivalis Strain A7A1-28.Human neutrophils and oral microbiota: a constant tug-of-war between a harmonious and a discordant coexistence.The inflammasome and danger molecule signaling: at the crossroads of inflammation and pathogen persistence in the oral cavity.Identification of Streptococcus cristatus peptides that repress expression of virulence genes in Porphyromonas gingivalis.Differential Regulation of Mas-Related G Protein-Coupled Receptor X2-Mediated Mast Cell Degranulation by Antimicrobial Host Defense Peptides and Porphyromonas gingivalis Lipopolysaccharide.Marginal bone loss around non-submerged implants is associated with salivary microbiome during bone healing
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P2860
Breaking bad: manipulation of the host response by Porphyromonas gingivalis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 2014
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Breaking bad: manipulation of the host response by Porphyromonas gingivalis.
@en
Breaking bad: manipulation of the host response by Porphyromonas gingivalis.
@nl
type
label
Breaking bad: manipulation of the host response by Porphyromonas gingivalis.
@en
Breaking bad: manipulation of the host response by Porphyromonas gingivalis.
@nl
prefLabel
Breaking bad: manipulation of the host response by Porphyromonas gingivalis.
@en
Breaking bad: manipulation of the host response by Porphyromonas gingivalis.
@nl
P2860
P356
P1476
Breaking bad: manipulation of the host response by Porphyromonas gingivalis
@en
P2093
Richard J Lamont
P2860
P304
P356
10.1002/EJI.201344202
P407
P577
2014-02-01T00:00:00Z