A systematic review of experimental treatments for mitochondrial dysfunction in sepsis and multiple organ dysfunction syndrome.
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Experimental treatments for mitochondrial dysfunction in sepsis: A narrative reviewHepatic mitochondrial function analysis using needle liver biopsy samplesDissecting inflammatory complications in critically injured patients by within-patient gene expression changes: a longitudinal clinical genomics study.Energy crisis: the role of oxidative phosphorylation in acute inflammation and sepsisLactate as a hemodynamic marker in the critically illCobalt protoporphyrin accelerates TFEB activation and lysosome reformation during LPS-induced septic insults in the rat heartValue of caffeic acid phenethyl ester pretreatment in experimental sepsis model in rats.Mitochondrial proteomes of porcine kidney cortex and medulla: foundation for translational proteomics.Coenzyme Q10 levels are low and may be associated with the inflammatory cascade in septic shock.Extrusion of mitochondrial contents from lipopolysaccharide-stimulated cells: Involvement of autophagy.The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology.Early alterations in platelet mitochondrial function are associated with survival and organ failure in patients with septic shock.Correlation of the oxygen radical activity and antioxidants and severity in critically ill surgical patients - study protocol.Coenzyme Q10 levels are low and associated with increased mortality in post-cardiac arrest patientsHeat shock protein 72 expressing stress in sepsis: unbridgeable gap between animal and human studies--a hypothetical "comparative" studyBench-to-bedside review: sepsis - from the redox point of view.Pharmacological targets in the renal peritubular microenvironment: implications for therapy for sepsis-induced acute kidney injury.Evidence to support mitochondrial neuroprotection, in severe traumatic brain injury.Integrative model of leukocyte genomics and organ dysfunction in heart failure patients requiring mechanical circulatory support: a prospective observational study.Multifactorial theory applied to the neurotoxicity of paraquat and paraquat-induced mechanisms of developing Parkinson's disease.Monitoring Severity of Multiple Organ Dysfunction Syndrome: New Technologies.The immune system's role in sepsis progression, resolution, and long-term outcome.Saturated hydrogen saline ameliorates lipopolysaccharide-induced acute lung injury by reducing excessive autophagy.Melatonin improved rat cardiac mitochondria and survival rate in septic heart injury.Broad defects in the energy metabolism of leukocytes underlie immunoparalysis in sepsis.N-acetylcysteine plus deferoxamine for patients with prolonged hypotension does not decrease acute kidney injury incidence: a double blind, randomized, placebo-controlled trial.Role of protein farnesylation in burn-induced metabolic derangements and insulin resistance in mouse skeletal muscle.A combination of 3D-QSAR, molecular docking and molecular dynamics simulation studies of benzimidazole-quinolinone derivatives as iNOS inhibitors.Elimination and active extrusion of liver mitochondrial proteins during lipopolysaccharide administration in rat.Early organ-specific mitochondrial dysfunction of jejunum and lung found in rats with experimental acute pancreatitisMitochondrial protection by the thioredoxin-2 and glutathione systems in an in vitro endothelial model of sepsis.Skeletal Muscle and Lymphocyte Mitochondrial Dysfunctions in Septic Shock Trigger ICU-Acquired Weakness and Sepsis-Induced Immunoparalysis.Multiscale measurement of cardiac energetics.Effect of acute pancreatitis on porcine intestine: a morphological study.Whole Blood Redox Potential Correlates with Progressive Accumulation of Oxygen Debt and Acts as a Marker of Resuscitation in a Swine Hemorrhagic Shock Model.Early septic shock induces loss of oxidative phosphorylation yield plasticity in liver mitochondria.Distinct Myocardial Mechanisms Underlie Cardiac Dysfunction in Endotoxemic Male and Female Mice.Nitrite administration improves sepsis-induced myocardial and mitochondrial dysfunction by modulating stress signal responses.Kir6.2 knockout aggravates lipopolysaccharide-induced mouse liver injury via enhancing NLRP3 inflammasome activation.Mitochondrial dysfunction in steatotic rat livers occurs because a defect in complex i makes the liver susceptible to prolonged cold ischemia.
P2860
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P2860
A systematic review of experimental treatments for mitochondrial dysfunction in sepsis and multiple organ dysfunction syndrome.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 26 August 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
A systematic review of experim ...... le organ dysfunction syndrome.
@en
A systematic review of experim ...... le organ dysfunction syndrome.
@nl
type
label
A systematic review of experim ...... le organ dysfunction syndrome.
@en
A systematic review of experim ...... le organ dysfunction syndrome.
@nl
prefLabel
A systematic review of experim ...... le organ dysfunction syndrome.
@en
A systematic review of experim ...... le organ dysfunction syndrome.
@nl
P2093
P1476
A systematic review of experim ...... ple organ dysfunction syndrome
@en
P2093
Anna J Dare
Anthony J R Hickey
Anthony R J Phillips
Anubhav Mittal
Benjamin Loveday
John A Windsor
P304
P356
10.1016/J.FREERADBIOMED.2009.08.019
P577
2009-08-26T00:00:00Z