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Nonradiographic axial spondyloarthritis: clinical and therapeutic relevance.Bone Disease in Axial Spondyloarthritis.Whodunit? The Contribution of Interleukin (IL)-17/IL-22-Producing γδ T Cells, αβ T Cells, and Innate Lymphoid Cells to the Pathogenesis of Spondyloarthritis.Genetically determined high activities of the TNF-alpha, IL23/IL17, and NFkB pathways were associated with increased risk of ankylosing spondylitisThe role of secukinumab in the treatment of psoriatic arthritis and ankylosing spondylitis
P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 18 January 2017
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Anti-IL17A in Axial Spondyloarthritis-Where Are We At?
@en
Anti-IL17A in Axial Spondyloarthritis-Where Are We At?
@nl
type
label
Anti-IL17A in Axial Spondyloarthritis-Where Are We At?
@en
Anti-IL17A in Axial Spondyloarthritis-Where Are We At?
@nl
prefLabel
Anti-IL17A in Axial Spondyloarthritis-Where Are We At?
@en
Anti-IL17A in Axial Spondyloarthritis-Where Are We At?
@nl
P2860
P356
P1476
Anti-IL17A in Axial Spondyloarthritis-Where Are We At?
@en
P2093
Peter P Cheung
P2860
P356
10.3389/FMED.2017.00001
P577
2017-01-18T00:00:00Z