Association of CD4+ CD25+ T cells with prevention of severe destructive arthritis in Borrelia burgdorferi-vaccinated and challenged gamma interferon-deficient mice treated with anti-interleukin-17 antibody.
about
TGF-β1 of no avail as prognostic marker in lyme diseaseThe challenge of assessing infant vaccine responses in resource-poor settingsClinical features and expressions of Foxp3 and IL-17 in type 1 autoimmune pancreatitis in ChinaAnti-CD25 antibody treatment of mice vaccinated and challenged with Borrelia spp. does not exacerbate arthritis but inhibits borreliacidal antibody production.Infection of Interleukin 17 Receptor A-Deficient C3H Mice with Borrelia burgdorferi Does Not Affect Their Development of Lyme Arthritis and Carditis.Anti-p19 antibody treatment exacerbates lyme arthritis and enhances borreliacidal activity.Th17 cells in human disease.Maintaining immunological tolerance with Foxp3.Immunological decision-making: how does the immune system decide to mount a helper T-cell response?Lyme arthritis: current concepts and a change in paradigm.Borrelia species induce inflammasome activation and IL-17 production through a caspase-1-dependent mechanismReduction of IL-17A might suppress the Th1 response and promote the Th2 response by boosting the function of Treg cells during silica-induced inflammatory response in vitro.Hamster and murine models of severe destructive Lyme arthritis.T-Helper 17 Cell Cytokine Responses in Lyme Disease Correlate With Borrelia burgdorferi Antibodies During Early Infection and With Autoantibodies Late in the Illness in Patients With Antibiotic-Refractory Lyme Arthritis.CD4+ cell-derived interleukin-17 in a model of dysregulated, Borrelia-induced arthritis.Arthritis is developed in Borrelia-primed and -infected mice deficient of interleukin-17.CD4+ lymphocytes and gamma interferon predominate in local immune responses in early experimental syphilis.CD4(+) CD25(+) T cells prevent arthritis associated with Borrelia vaccination and infectionInterleukin-35 enhances Lyme arthritis in Borrelia-vaccinated and -infected mice.Regulatory T cells, a potent immunoregulatory target for CAM researchers: modulating tumor immunity, autoimmunity and alloreactive immunity (III).Interleukin-23 is required for development of arthritis in mice vaccinated and challenged with Borrelia species.Anti-interleukin-15 prevents arthritis in Borrelia-vaccinated and -infected miceInterleukin-10 (IL-10) inhibits Borrelia burgdorferi-induced IL-17 production and attenuates IL-17-mediated Lyme arthritis.
P2860
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P2860
Association of CD4+ CD25+ T cells with prevention of severe destructive arthritis in Borrelia burgdorferi-vaccinated and challenged gamma interferon-deficient mice treated with anti-interleukin-17 antibody.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Association of CD4+ CD25+ T ce ...... anti-interleukin-17 antibody.
@en
Association of CD4+ CD25+ T ce ...... anti-interleukin-17 antibody.
@nl
type
label
Association of CD4+ CD25+ T ce ...... anti-interleukin-17 antibody.
@en
Association of CD4+ CD25+ T ce ...... anti-interleukin-17 antibody.
@nl
prefLabel
Association of CD4+ CD25+ T ce ...... anti-interleukin-17 antibody.
@en
Association of CD4+ CD25+ T ce ...... anti-interleukin-17 antibody.
@nl
P2093
P2860
P1476
Association of CD4+ CD25+ T ce ...... anti-interleukin-17 antibody.
@en
P2093
Dean T Nardelli
Douglas M England
Jose Torrealba
Matthew A Burchill
Ronald F Schell
Steven M Callister
P2860
P304
P356
10.1128/CDLI.11.6.1075-1084.2004
P577
2004-11-01T00:00:00Z