The role of vimentin intermediate filaments in the progression of lung cancer.
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Intermediate Filaments as Organizers of Cellular Space: How They Affect Mitochondrial Structure and FunctionClinical impacts of a micropapillary pattern in lung adenocarcinoma: a reviewSignificance of circulating tumor cells in soft tissue sarcoma.From Proteomic Analysis to Potential Therapeutic Targets: Functional Profile of Two Lung Cancer Cell Lines, A549 and SW900, Widely Studied in Pre-Clinical ResearchMethods for Determining the Cellular Functions of Vimentin Intermediate FilamentsVimentin is a potential prognostic factor for tongue squamous cell carcinoma among five epithelial-mesenchymal transition-related proteins.KLF4 Plays an Essential Role in Corneal Epithelial Homeostasis by Promoting Epithelial Cell Fate and Suppressing Epithelial-Mesenchymal Transition.Clear Cell Renal Cell Carcinoma is linked to Epithelial-to-Mesenchymal Transition and to FibrosisURI promotes the migration and invasion of human cervical cancer cells potentially via upregulation of vimentin expression.Quantitative Proteomics Analysis Reveals Novel Insights into Mechanisms of Action of Long Noncoding RNA Hox Transcript Antisense Intergenic RNA (HOTAIR) in HeLa CellsPrognostic Values of Vimentin Expression and Its Clinicopathological Significance in Non-Small Cell Lung Cancer: A Meta-Analysis of Observational Studies with 4118 Cases.Vimentin regulates differentiation switch via modulation of keratin 14 levels and their expression together correlates with poor prognosis in oral cancer patients.Metformin inhibits gastric cancer cells metastatic traits through suppression of epithelial-mesenchymal transition in a glucose-independent manner.Differentially expressed proteins in glioblastoma multiforme identified with a nanobody-based anti-proteome approach and confirmed by OncoFinder as possible tumor-class predictive biomarker candidates.Expression of pituitary tumor-transforming 2 in human glioblastoma cell lines and its role in glioblastoma tumorigenesisA Multiplexed Cytokeratin Analysis Using Targeted Mass Spectrometry Reveals Specific Profiles in Cancer-Related Pleural EffusionsRac1-mediated cytoskeleton rearrangements induced by intersectin-1s deficiency promotes lung cancer cell proliferation, migration and metastasis.Beta-catenin participates in dialysate-induced peritoneal fibrosis via enhanced peritoneal cell epithelial-to-mesenchymal transition.Cytoskeleton-centric protein transportation by exosomes transforms tumor-favorable macrophages.Aptamers Selected to Postoperative Lung Adenocarcinoma Detect Circulating Tumor Cells in Human Blood.Vimentin Intermediate Filaments Template Microtubule Networks to Enhance Persistence in Cell Polarity and Directed Migration.Intermediate filament proteins of digestive organs: physiology and pathophysiology.Structural Dynamics of the Vimentin Coiled-coil Contact Regions Involved in Filament Assembly as Revealed by Hydrogen-Deuterium Exchange.Epithelial-Mesenchymal Transition Protein Expression in Basal Cell Adenomas and Basal Cell Adenocarcinomas.Lung cancer exosomes as drivers of epithelial mesenchymal transition.Resistance to epithelial growth factor receptor tyrosine kinase inhibitors in a patient with transformation from lung adenocarcinoma to small cell lung cancer: A case report.Retinoblastoma protein (Rb) links hypoxia to altered mechanical properties in cancer cells as measured by an optical tweezer.USP14 de-ubiquitinates vimentin and miR-320a modulates USP14 and vimentin to contribute to malignancy in gastric cancer cells.DNA Aptamers for the Characterization of Histological Structure of Lung Adenocarcinoma.Correlation of MMP-9 and p53 protein expression with prognosis in metastatic spinal tumor of lung cancer.CBP501 inhibits EGF-dependent cell migration, invasion and epithelial-to-mesenchymal transition of non-small cell lung cancer cells by blocking KRas to calmodulin binding.Integrative Temporo-Spatial, Mineralogic, Spectroscopic, and Proteomic Analysis of Postnatal Enamel Development in Teeth with Limited Growth.Current and Prospective Protein Biomarkers of Lung Cancer.Human Primary Epithelial Cells Acquire an Epithelial-Mesenchymal-Transition Phenotype during Long-Term Infection by the Oral Opportunistic Pathogen, Porphyromonas gingivalis.14-3-3 proteins: an important regulator of autophagy in diseases.Type III Intermediate Filaments Desmin, Glial Fibrillary Acidic Protein (GFAP), Vimentin, and Peripherin.Checkpoint kinase 1-induced phosphorylation of O-linked β-N-acetylglucosamine transferase regulates the intermediate filament network during cytokinesis.Intermediate Filaments and the Regulation of Cell Motility during Regeneration and Wound Healing.Conditioned Medium from Adipose-Derived Stem Cells (ADSCs) Promotes Epithelial-to-Mesenchymal-Like Transition (EMT-Like) in Glioma Cells In vitro.MiR-378 and MiR-1827 Regulate Tumor Invasion, Migration and Angiogenesis in Human Lung Adenocarcinoma by Targeting RBX1 and CRKL, Respectively.
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The role of vimentin intermediate filaments in the progression of lung cancer.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on January 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The role of vimentin intermediate filaments in the progression of lung cancer.
@en
The role of vimentin intermediate filaments in the progression of lung cancer.
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type
label
The role of vimentin intermediate filaments in the progression of lung cancer.
@en
The role of vimentin intermediate filaments in the progression of lung cancer.
@nl
prefLabel
The role of vimentin intermediate filaments in the progression of lung cancer.
@en
The role of vimentin intermediate filaments in the progression of lung cancer.
@nl
P2093
P2860
P356
P1476
The role of vimentin intermediate filaments in the progression of lung cancer.
@en
P2093
Dale K Shumaker
Karen M Ridge
Martha E Kidd
P2860
P356
10.1165/RCMB.2013-0314TR
P577
2014-01-01T00:00:00Z