Subcellular and metabolic examination of amyloid-beta peptides in Alzheimer disease pathogenesis: evidence for Abeta(25-35).
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Calcium-Sensing Receptors of Human Neural Cells Play Crucial Roles in Alzheimer's DiseaseIn Vivo Characterization of ARN14140, a Memantine/Galantamine-Based Multi-Target Compound for Alzheimer's DiseaseEstradiol prevents olfactory dysfunction induced by A-β 25-35 injection in hippocampus.Arrestins contribute to amyloid beta-induced cell death via modulation of autophagy and the α7nAch receptor in SH-SY5Y cells.Structural determination of Abeta25-35 micelles by molecular dynamics simulations.Neuroprotection by the synthetic neurosteroid enantiomers ent-PREGS and ent-DHEAS against Aβ₂₅₋₃₅ peptide-induced toxicity in vitro and in vivo in miceCalcium-Sensing Receptors of Human Astrocyte-Neuron Teams: Amyloid-β-Driven Mediators and Therapeutic Targets of Alzheimer's DiseaseBrain-derived neurotrophic factor exerts neuroprotective actions against amyloid β-induced apoptosis in neuroblastoma cells.Mulberry extracts alleviate aβ 25-35-induced injury and change the gene expression profile in PC12 cells.Amyloid-β as a modulator of synaptic plasticityPARP-1 modulates amyloid beta peptide-induced neuronal damage.Frontiers in Alzheimer's disease therapeutics.When one plus one does not equal two: fluorescence anisotropy in aggregates and multiply labeled proteins.Role of amyloid peptides in vascular dysfunction and platelet dysregulation in Alzheimer's diseaseInhibition of the polyamine system counteracts β-amyloid peptide-induced memory impairment in mice: involvement of extrasynaptic NMDA receptors.Interaction of Aβ(25-35) fibrillation products with mitochondria: Effect of small-molecule natural products.TNFAIP1 contributes to the neurotoxicity induced by Aβ25-35 in Neuro2a cells.Role of β-hairpin formation in aggregation: the self-assembly of the amyloid-β(25-35) peptide.Na+ and K+ ion imbalances in Alzheimer's disease.The protection of acetylcholinesterase inhibitor on β-amyloid-induced the injury of neurite outgrowth via regulating axon guidance related genes expression in neuronal cells.Neuroprotective effects of sodium hydrosulfide against β-amyloid-induced neurotoxicity.Combining two repurposed drugs as a promising approach for Alzheimer's disease therapyAn isoform-specific role of FynT tyrosine kinase in Alzheimer's disease.Genetics of β-Amyloid Precursor Protein in Alzheimer's Disease.14-3-3ε marks the amyloid-stimulated microglia long-term activation.Inhibition of amyloid peptide fragment Aβ25-35 fibrillogenesis and toxicity by N-terminal β-amino acid-containing esapeptides: is taurine moiety essential for in vivo effects?Aβ25-35 Suppresses Mitochondrial Biogenesis in Primary Hippocampal Neurons.β-asarone prevents Aβ25-35-induced inflammatory responses and autophagy in SH-SY5Y cells: down expression Beclin-1, LC3B and up expression Bcl-2.Structure-dependent effects of amyloid-β on long-term memory in Lymnaea stagnalis.Complexing Aβ prevents the cellular anomalies induced by the Peptide alone.Amyloid and Alzheimer's disease.Amyloid β-peptide-dependent activation of human platelets: essential role for Ca2+ and ADP in aggregation and thrombus formation.Non-aggregated Aβ25-35 Upregulates Primary Astrocyte Proliferation In Vitro.Amyloid β-peptide 1-42 modulates the proliferation of mouse neural stem cells: upregulation of fucosyltransferase IX and notch signaling.Regulation of adenosine triphosphate-sensitive potassium channels suppresses the toxic effects of amyloid-beta peptide (25-35)Influence of Au nanoparticles on the aggregation of amyloid-β-(25-35) peptides.In vivo and ex vivo analyses of amyloid toxicity in the Tc1 mouse model of Down syndrome.Effects of Acetylcholine on β-Amyloid-Induced cPLA2 Activation in the TB Neuroectodermal Cell Line: Implications for the Pathogenesis of Alzheimer's Disease.Metabolic Abnormalities of Erythrocytes as a Risk Factor for Alzheimer's Disease.Tanshinone IIA attenuates Aβ25-35 -induced spatial memory impairment via upregulating receptors for activated C kinase1 and inhibiting autophagy in hippocampus.
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Subcellular and metabolic examination of amyloid-beta peptides in Alzheimer disease pathogenesis: evidence for Abeta(25-35).
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 12 September 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Subcellular and metabolic exam ...... thogenesis: evidence for Abeta
@nl
Subcellular and metabolic exam ...... is: evidence for Abeta(25-35).
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type
label
Subcellular and metabolic exam ...... thogenesis: evidence for Abeta
@nl
Subcellular and metabolic exam ...... is: evidence for Abeta(25-35).
@en
prefLabel
Subcellular and metabolic exam ...... thogenesis: evidence for Abeta
@nl
Subcellular and metabolic exam ...... is: evidence for Abeta(25-35).
@en
P2093
P1476
Subcellular and metabolic exam ...... is: evidence for Abeta(25-35).
@en
P2093
Elena A Kosenko
Michael W Marlatt
Yury G Kaminsky
P356
10.1016/J.EXPNEUROL.2009.09.005
P407
P577
2009-09-12T00:00:00Z