Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy. - Wikidata - awgldk - TriplyDB

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

http://www.wikidata.org/entity/Q37600956

about

A novel ubiquitin-binding protein ZNF216 functioning in muscle atrophy PC4/Tis7/IFRD1 stimulates skeletal muscle regeneration and is involved in myoblast differentiation as a regulator of MyoD and NF-kappaB Effects of dietary curcumin or N-acetylcysteine on NF-kappaB activity and contractile performance in ambulatory and unloaded murine soleus Cellular and molecular mechanisms of muscle atrophy Longevity and skeletal muscle mass: the role of IGF signalling, the sirtuins, dietary restriction and protein intake Oxygen consumption and usage during physical exercise: the balance between oxidative stress and ROS-dependent adaptive signaling Tumor necrosis factor-α regulates distinct molecular pathways and gene networks in cultured skeletal muscle cells REDD1 is a major target of testosterone action in preventing dexamethasone-induced muscle loss p38 MAPK links oxidative stress to autophagy-related gene expression in cachectic muscle wasting.The Systemic Effect of Burn Injury and Trauma on Muscle and Bone Mass and Composition.NF-kappaB mediates the transcription of mouse calsarcin-1 gene, but not calsarcin-2, in C2C12 cells Dynamic resolution of functionally related gene sets in response to acute heat stress Role of proteasomes in disease.Limb immobilization induces a coordinate down-regulation of mitochondrial and other metabolic pathways in men and women Genomic profiling of messenger RNAs and microRNAs reveals potential mechanisms of TWEAK-induced skeletal muscle wasting in mice The mERG1a channel modulates skeletal muscle MuRF1, but not MAFbx, expression.The TWEAK-Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice.Oxidative stress, molecular inflammation and sarcopenia The proteasome inhibitor MG132 reduces immobilization-induced skeletal muscle atrophy in mice TWEAK causes myotube atrophy through coordinated activation of ubiquitin-proteasome system, autophagy, and caspases.TNF-related weak inducer of apoptosis (TWEAK) is a potent skeletal muscle-wasting cytokine Characterization of GLPG0492, a selective androgen receptor modulator, in a mouse model of hindlimb immobilization NF-κB activity in muscle from obese and type 2 diabetic subjects under basal and exercise-stimulated conditions.Identification of genes that elicit disuse muscle atrophy via the transcription factors p50 and Bcl-3.The ChIP-seq-defined networks of Bcl-3 gene binding support its required role in skeletal muscle atrophy.Nuclear factor-kappa B signaling in skeletal muscle atrophy.Bcl3 interacts cooperatively with peroxisome proliferator-activated receptor gamma (PPARgamma) coactivator 1alpha to coactivate nuclear receptors estrogen-related receptor alpha and PPARalpha Inhibition of IkappaB kinase alpha (IKKα) or IKKbeta (IKKβ) plus forkhead box O (Foxo) abolishes skeletal muscle atrophy.Neurofibromin (Nf1) is required for skeletal muscle development C26 cancer-induced muscle wasting is IKKβ-dependent and NF-kappaB-independent.Expression of NF-kappaB and IkappaB proteins in skeletal muscle of gastric cancer patients.Central nervous system inflammation induces muscle atrophy via activation of the hypothalamic-pituitary-adrenal axis.The TWEAK-Fn14 system: breaking the silence of cytokine-induced skeletal muscle wasting.Interplay of IKK/NF-kappaB signaling in macrophages and myofibers promotes muscle degeneration in Duchenne muscular dystrophy.NF-κB in Aging and Disease Molecular events and signalling pathways involved in skeletal muscle disuse-induced atrophy and the impact of countermeasures.NO production results in suspension-induced muscle atrophy through dislocation of neuronal NOS.RNA-Seq Profiling of Intact and Enucleated Oocyte SCNT Embryos Reveals the Role of Pig Oocyte Nucleus in Somatic Reprogramming.Lack of NF-kappaB1 (p105/p50) attenuates unloading-induced downregulation of PPARalpha and PPARalpha-regulated gene expression in rodent heart.Transcriptome profile of rat genes in injured spinal cord at different stages by RNA-sequencing.

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P2860

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

http://www.wikidata.org/entity/Q37600956

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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name

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

@en

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

@nl

type

label

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

@en

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

@nl

prefLabel

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

@en

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

@nl

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Journal of Clinical Investigation

P1476

Disruption of either the Nfkb1 or the Bcl3 gene inhibits skeletal muscle atrophy.

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R Bridge Hunter

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Susan C Kandarian

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P2860

The Bcl-3 oncoprotein acts as a bridging factor between NF-kappaB/Rel and nuclear co-regulators

Regulation of NFKB1 proteins by the candidate oncoprotein BCL-3: generation of NF-kappaB homodimers from the cytoplasmic pool of p50-p105 and nuclear translocation.

NF-kappaB p105 is a target of IkappaB kinases and controls signal induction of Bcl-3-p50 complexes

Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy

Characterization of antigen receptor response elements within the interleukin-2 enhancer

The ubiquitin system

NF-kappa B and Rel proteins: evolutionarily conserved mediators of immune responses

Muscle cachexia: current concepts of intracellular mechanisms and molecular regulation

The candidate oncoprotein Bcl-3 is an antagonist of p50/NF-kappa B-mediated inhibition

Activation of nuclear factor-kappaB p50 homodimer/Bcl-3 complexes in nasopharyngeal carcinoma

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1504-1511

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10.1172/JCI21696

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