B cells promote induction of experimental autoimmune encephalomyelitis by facilitating reactivation of T cells in the central nervous system.
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B-cell depletion attenuates serological biomarkers of fibrosis and myofibroblast activation in IgG4-related diseaseB Cells Are Multifunctional Players in Multiple Sclerosis Pathogenesis: Insights from Therapeutic InterventionsCD8(+) T-Cells as Immune Regulators of Multiple SclerosisNovel Insights and Therapeutics in Multiple SclerosisB Cells and Autoantibodies in Multiple SclerosisRole of regulatory b cells in neuroimmunologic disordersMultiple Sclerosis and T Lymphocytes: An Entangled StoryRelapsing-remitting central nervous system autoimmunity mediated by GFAP-specific CD8 T cells.Progression from IgD+ IgM+ to isotype-switched B cells is site specific during coronavirus-induced encephalomyelitis.Categorization of multiple sclerosis relapse subtypes by B cell profiling in the bloodB-cell very late antigen-4 deficiency reduces leukocyte recruitment and susceptibility to central nervous system autoimmunity.Myelin Basic Protein-Induced Production of Tumor Necrosis Factor-α and Interleukin-6, and Presentation of the Immunodominant Peptide MBP85-99 by B Cells from Patients with Relapsing-Remitting Multiple Sclerosis.Rituximab in Children with Steroid-Dependent Nephrotic Syndrome: A Multicenter, Open-Label, Noninferiority, Randomized Controlled Trial.Determining Immune System Suppression versus CNS Protection for Pharmacological Interventions in Autoimmune Demyelination.MicroRNAs: Roles in Regulating Neuroinflammation.Regulatory T cells and minimal change nephropathy: in the midst of a complex network.Illuminating vitamin D effects on B cells--the multiple sclerosis perspective.Influenza infection triggers disease in a genetic model of experimental autoimmune encephalomyelitis.Potential importance of B cells in aging and aging-associated neurodegenerative diseases.Mechanisms by Which B Cells and Regulatory T Cells Influence Development of Murine Organ-Specific Autoimmune DiseasesActivated GL7+ B cells are maintained within the inflamed CNS in the absence of follicle formation during viral encephalomyelitis.B cells are capable of independently eliciting rapid reactivation of encephalitogenic CD4 T cells in a murine model of multiple sclerosis.
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P2860
B cells promote induction of experimental autoimmune encephalomyelitis by facilitating reactivation of T cells in the central nervous system.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 23 December 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
B cells promote induction of e ...... in the central nervous system.
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B cells promote induction of e ...... in the central nervous system.
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B cells promote induction of e ...... in the central nervous system.
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B cells promote induction of e ...... in the central nervous system.
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B cells promote induction of e ...... in the central nervous system.
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B cells promote induction of e ...... in the central nervous system.
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P2860
P356
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B cells promote induction of e ...... in the central nervous system.
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Emily R Pierson
Ingunn M Stromnes
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P304
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10.4049/JIMMUNOL.1302171
P407
P577
2013-12-23T00:00:00Z