CXCL5-secreting pulmonary epithelial cells drive destructive neutrophilic inflammation in tuberculosis. - Wikidata - awgldk - TriplyDB

CXCL5-secreting pulmonary epithelial cells drive destructive neutrophilic inflammation in tuberculosis.

CXCL5-secreting pulmonary epithelial cells drive destructive neutrophilic inflammation in tuberculosis.

http://www.wikidata.org/entity/Q37602224

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Innate Immune Defenses in Human Tuberculosis: An Overview of the Interactions between Mycobacterium tuberculosis and Innate Immune Cells Versatile myeloid cell subsets contribute to tuberculosis-associated inflammation Chemokine receptors and their therapeutic opportunities in diseased lung: far beyond leukocyte trafficking Reverse translation in tuberculosis: neutrophils provide clues for understanding development of active disease Granulomas and Inflammation: Host-Directed Therapies for Tuberculosis.Dissection of the host-pathogen interaction in human tuberculosis using a bioengineered 3-dimensional model Complex regulation of neutrophil-derived MMP-9 secretion in central nervous system tuberculosis Influenza A induces dysfunctional immunity and death in MeCP2-overexpressing mice.Neutrophils in Tuberculosis: Heterogeneity Shapes the Way?Myeloid derived hypoxia inducible factor 1-alpha is required for protection against pulmonary Aspergillus fumigatus infection Beyond macrophages: the diversity of mononuclear cells in tuberculosis.Platelets direct monocyte differentiation into epithelioid-like multinucleated giant foam cells with suppressive capacity upon mycobacterial stimulation.CXCL5 drives neutrophil recruitment in TH17-mediated GN Type I IFN signaling triggers immunopathology in tuberculosis-susceptible mice by modulating lung phagocyte dynamics.Neutrophil-Derived MMP-8 Drives AMPK-Dependent Matrix Destruction in Human Pulmonary Tuberculosis The microbiome at the pulmonary alveolar niche and its role in Mycobacterium tuberculosis infection Bacillary replication and macrophage necrosis are determinants of neutrophil recruitment in tuberculosis.IL-17RA in Non-Hematopoietic Cells Controls CXCL-1 and 5 Critical to Recruit Neutrophils to the Lung of Mycobacteria-Infected Mice during the Adaptive Immune Response The Src-Family Kinases Hck and Fgr Regulate Early Lipopolysaccharide-Induced Myeloid Cell Recruitment into the Lung and Their Ability To Secrete Chemokines.An expansion of rare lineage intestinal microbes characterizes rheumatoid arthritis.Lung necrosis and neutrophils reflect common pathways of susceptibility to Mycobacterium tuberculosis in genetically diverse, immune-competent mice.Helminth-induced arginase-1 exacerbates lung inflammation and disease severity in tuberculosis Alveolar Epithelial Cells in Mycobacterium tuberculosis Infection: Active Players or Innocent Bystanders?Mouse models of human TB pathology: roles in the analysis of necrosis and the development of host-directed therapies.No involvement of alveolar macrophages in the initiation of carbon nanoparticle induced acute lung inflammation in mice.Curcumin suppresses NTHi-induced CXCL5 expression via inhibition of positive IKKβ pathway and up-regulation of negative MKP-1 pathway.Pseudomonas aeruginosa infection augments inflammation through miR-301b repression of c-Myb-mediated immune activation and infiltration.Mucosal BCG Vaccination Induces Protective Lung-Resident Memory T Cell Populations against Tuberculosis.Chemokines in tuberculosis: the good, the bad and the ugly.The onset of adaptive immunity in the mouse model of tuberculosis and the factors that compromise its expression.Mycobacterium tuberculosis infection of the 'non-classical immune cell'.Host-Directed Therapies for Tuberculosis.Nitric oxide prevents a pathogen-permissive granulocytic inflammation during tuberculosis.Understanding the pathophysiology of the human TB lung granuloma using in vitro granuloma models.Toll-like receptor 2 in host defense against Mycobacterium tuberculosis: to be or not to be-that is the question.Transcriptional Regulation of CXCL5 in HIV-1-Infected Macrophages and Its Functional Consequences on CNS Pathology.Cytokines and Chemokines in Mycobacterium tuberculosis Infection.IL-17 Receptor Signaling in the Lung Epithelium Is Required for Mucosal Chemokine Gradients and Pulmonary Host Defense against K. pneumoniae.Multiplicity of Mathematical Modeling Strategies to Search for Molecular and Cellular Insights into Bacteria Lung Infection.Innate immunity in tuberculosis: host defense vs pathogen evasion.

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CXCL5-secreting pulmonary epithelial cells drive destructive neutrophilic inflammation in tuberculosis.

http://www.wikidata.org/entity/Q37602224

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 10 February 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

CXCL5-secreting pulmonary epit ...... inflammation in tuberculosis.

@en

CXCL5-secreting pulmonary epit ...... inflammation in tuberculosis.

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type

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CXCL5-secreting pulmonary epit ...... inflammation in tuberculosis.

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CXCL5-secreting pulmonary epit ...... inflammation in tuberculosis.

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CXCL5-secreting pulmonary epit ...... inflammation in tuberculosis.

@en

CXCL5-secreting pulmonary epit ...... inflammation in tuberculosis.

@nl

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Journal of Clinical Investigation

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CXCL5-secreting pulmonary epit ...... inflammation in tuberculosis.

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Anca Dorhoi

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Catherine Meyer-Schwesinger

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Delia Loewe

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Gayle McEwen

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Geraldine Nouailles

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Hans-Willi Mittrücker

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January Weiner

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Jens Zerrahn

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Kellen C Faé

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Markus Koch

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Induction of direct antimicrobial activity through mammalian toll-like receptors

Chemokine production by a human alveolar epithelial cell line in response to Mycobacterium tuberculosis

CXCR5⁺ T helper cells mediate protective immunity against tuberculosis

The chemokine superfamily revisited

Mycobacterium tuberculosis: success through dormancy

Linear models and empirical bayes methods for assessing differential expression in microarray experiments

A new mathematical model for relative quantification in real-time RT-PCR

Neutrophil and B cell expansion in mice that lack the murine IL-8 receptor homolog

Cxcr2 and Cxcl5 regulate the IL-17/G-CSF axis and neutrophil homeostasis in mice

CXCL5 limits macrophage foam cell formation in atherosclerosis

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1268-1282

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scholarly article

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10.1172/JCI72030

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3

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124

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Stefan Kaufmann

Hans-Willi Mittrücker

Hans-Joachim Mollenkopf

Alexis Vogelzang

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2014-02-10T00:00:00Z

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24509076

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Akafuba bulwadde

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3934185

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