Mifepristone decreases depression-like behavior and modulates neuroendocrine and central hypothalamic-pituitary-adrenocortical axis responsiveness to stress.
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Coping with the Forced Swim Stressor: Towards Understanding an Adaptive MechanismAdolescent chronic stress causes hypothalamo-pituitary-adrenocortical hypo-responsiveness and depression-like behavior in adult female rats.Mifepristone modulates serotonin transporter functionAntidepressant-Like Effects of Lindera obtusiloba Extracts on the Immobility Behavior of Rats in the Forced Swim TestHypothalamic-pituitary-adrenocortical axis dysfunction in epilepsy.Stress vulnerability during adolescent development in rats.Role of prefrontal cortex glucocorticoid receptors in stress and emotionAll-trans retinoic acid-induced hypothalamus-pituitary-adrenal hyperactivity involves glucocorticoid receptor dysregulation.A single-day treatment with mifepristone is sufficient to normalize chronic glucocorticoid induced suppression of hippocampal cell proliferation.Pattern of c-Fos expression induced by tail suspension test in the mouse brain.The selective glucocorticoid receptor antagonist CORT 108297 decreases neuroendocrine stress responses and immobility in the forced swim test.Glucocorticoid receptors in the nucleus of the solitary tract (NTS) decrease endocrine and behavioral stress responsesMifepristone pretreatment reduces ethanol withdrawal severity in vivo.Progesterone receptor antagonist CDB-4124 increases depression-like behavior in mice without affecting locomotor ability.Glucocorticoid receptors in the locus coeruleus mediate sleep disorders caused by repeated corticosterone treatment.The neurobiological correlates of childhood adversity and implications for treatment.Mifepristone in the central nucleus of the amygdala reduces yohimbine stress-induced reinstatement of ethanol-seekingAngelica gigas ameliorate depression-like symptoms in rats following chronic corticosterone injection.Neuroendocrine Function After Hypothalamic Depletion of Glucocorticoid Receptors in Male and Female Mice.Differences in FKBP51 regulation following chronic social defeat stress correlate with individual stress sensitivity: influence of paroxetine treatment.Bidirectional crosstalk between stress-induced gastric ulcer and depression under chronic stress.Molecular Neurobiology and Promising New Treatment in Depression.Differential targeting of brain stress circuits with a selective glucocorticoid receptor modulator.Restoration of neuroendocrine stress response by glucocorticoid receptor or GABA(A) receptor antagonists after experimental traumatic brain injuryThe role of the glucocorticoids in developing resilience to stress and addiction.Mifepristone Accelerates HPA Axis Recovery in Secondary Adrenal InsufficiencyRU486 Mitigates Hippocampal Pathology Following Status Epilepticus.Deletion of forebrain glucocorticoid receptors impairs neuroendocrine stress responses and induces depression-like behavior in males but not females.Could the underestimation of bipolarity obstruct the search for novel antidepressant drugs?Mifepristone: a potential clinical agent based on its anti-progesterone and anti-glucocorticoid properties.Genomic and Nongenomic Effects of Mifepristone at the Cardiovascular Level: A Review.HPA Axis Interactions with Behavioral Systems.A mixed glucocorticoid/mineralocorticoid receptor modulator dampens endocrine and hippocampal stress responsivity in male rats.Behavioral and physiological consequences of enrichment loss in ratsFootshock-induced plasticity of GABAB signalling in the lateral habenula requires dopamine and glucocorticoid receptors.Cannabinoid receptor activation prevents the effects of chronic mild stress on emotional learning and LTP in a rat model of depressionThe modulatory role of the lateral septum on neuroendocrine and behavioral stress responses.Decreased daytime illumination leads to anxiety-like behaviors and HPA axis dysregulation in the diurnal grass rat (Arvicanthis niloticus).Mifepristone treatment affects the response to repeated amphetamine injections, but does not attenuate the expression of sensitization.Adolescent environmental enrichment prevents behavioral and physiological sequelae of adolescent chronic stress in female (but not male) rats.
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P2860
Mifepristone decreases depression-like behavior and modulates neuroendocrine and central hypothalamic-pituitary-adrenocortical axis responsiveness to stress.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 10 February 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Mifepristone decreases depress ...... axis responsiveness to stress.
@en
Mifepristone decreases depress ...... axis responsiveness to stress.
@nl
type
label
Mifepristone decreases depress ...... axis responsiveness to stress.
@en
Mifepristone decreases depress ...... axis responsiveness to stress.
@nl
prefLabel
Mifepristone decreases depress ...... axis responsiveness to stress.
@en
Mifepristone decreases depress ...... axis responsiveness to stress.
@nl
P2860
P1476
Mifepristone decreases depress ...... axis responsiveness to stress.
@en
P2093
Aynara C Wulsin
Matia B Solomon
P2860
P304
P356
10.1016/J.PSYNEUEN.2010.01.011
P577
2010-02-10T00:00:00Z