Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes.
about
Pyk2 inhibition of p53 as an adaptive and intrinsic mechanism facilitating cell proliferation and survivalIntegrins and integrin-associated proteins in the cardiac myocyte.Cardiomyocyte-specific expression of CRNK, the C-terminal domain of PYK2, maintains ventricular function and slows ventricular remodeling in a mouse model of dilated cardiomyopathy.Pyk2 activation triggers epidermal growth factor receptor signaling and cell motility after wounding sheets of epithelial cells.
P2860
Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 12 May 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes.
@en
Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes.
@nl
type
label
Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes.
@en
Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes.
@nl
prefLabel
Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes.
@en
Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes.
@nl
P2860
P1476
Differential effects of Pyk2 and FAK on the hypertrophic response of cardiac myocytes.
@en
P2093
Emmanuel B Menashi
Joseph C Loftus
P2860
P2888
P304
P356
10.1007/S00441-009-0807-9
P577
2009-05-12T00:00:00Z