CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism. - Wikidata - awgldk - TriplyDB

CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

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Is There a Role for Genomics in the Management of Hypertension?Subtype Diagnosis of Primary Aldosteronism: Is Adrenal Vein Sampling Always Necessary?Activity-dependent regulation of T-type calcium channels by submembrane calcium ions Many Channels Lead to Aldosterone.Genetic mechanisms of human hypertension and their implications for blood pressure physiology.Genetic Programming of Hypertension.The Low-Renin Hypertension Phenotype: Genetics and the Role of the Mineralocorticoid Receptor.Functional variants in HCN4 and CACNA1H may contribute to genetic generalized epilepsy.Comparative Genomics and Transcriptome Profiling in Primary Aldosteronism.

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CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

http://www.wikidata.org/entity/Q37604722

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 04 October 2016

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

@en

CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

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type

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CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

@en

CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

@nl

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CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

@en

CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

@nl

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J.EBIOM.2016.10.002

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CACNA1H Mutations Are Associated With Different Forms of Primary Aldosteronism.

@en

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Georgios Daniil

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Iulia Blesneac

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Jacques Beltrand

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Michel Polak

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Philippe Lory

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Tim M Strom

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P2860

A novel genetic locus for low renin hypertension: familial hyperaldosteronism type II maps to chromosome 7 (7p22)

T-Type Calcium Channel: A Privileged Gate for Calcium Entry and Control of Adrenal Steroidogenesis

Association between genetic variation of CACNA1H and childhood absence epilepsy

Molecular physiology of low-voltage-activated t-type calcium channels

The MIQE guidelines: minimum information for publication of quantitative real-time PCR experiments

Gating effects of mutations in the Cav3.2 T-type calcium channel associated with childhood absence epilepsy

Effects of Cav3.2 channel mutations linked to idiopathic generalized epilepsy

Further evidence for linkage of familial hyperaldosteronism type II at chromosome 7p22 in Italian as well as Australian and South American families

The amino side of the C-terminus determines fast inactivation of the T-type calcium channel alpha1G

Inactivation determinants in segment IIIS6 of Ca(v)3.1

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225-236

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scholarly article

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10.1016/J.EBIOM.2016.10.002

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13

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Xavier Jeunemaitre

Sheerazed Boulkroun

Maria-Christina Zennaro

Fabio L Fernandes-Rosa

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2016-10-04T00:00:00Z

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27729216

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5264314

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