Potential of metabolic agents as adjunct therapies in heart failure. - Wikidata - awgldk - TriplyDB

Potential of metabolic agents as adjunct therapies in heart failure.

Potential of metabolic agents as adjunct therapies in heart failure.

http://www.wikidata.org/entity/Q37608595

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Left ventricular strain and untwist in hypertrophic cardiomyopathy: relation to exercise capacity.Early diastolic function during exertion influences exercise intolerance in patients with hypertrophic cardiomyopathy.The pathophysiology of diastolic heart failure

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Potential of metabolic agents as adjunct therapies in heart failure.

http://www.wikidata.org/entity/Q37608595

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

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bilimsel makale

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scientific article published on September 2007

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Potential of metabolic agents as adjunct therapies in heart failure.

@en

Potential of metabolic agents as adjunct therapies in heart failure.

@nl

type

label

Potential of metabolic agents as adjunct therapies in heart failure.

@en

Potential of metabolic agents as adjunct therapies in heart failure.

@nl

prefLabel

Potential of metabolic agents as adjunct therapies in heart failure.

@en

Potential of metabolic agents as adjunct therapies in heart failure.

@nl

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14796678.3.5.525

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Future Cardiology

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Potential of metabolic agents as adjunct therapies in heart failure.

@en

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Abdul R Maher

http://www.w3.org/2001/XMLSchema#string

Ganesh Nallur Shivu

http://www.w3.org/2001/XMLSchema#string

Ibrar Ahmed

http://www.w3.org/2001/XMLSchema#string

Khalid Abozguia

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Michael Frenneaux

http://www.w3.org/2001/XMLSchema#string

Thanh Trung Phan

http://www.w3.org/2001/XMLSchema#string

P2860

Phosphorylation and activation of heart PFK-2 by AMPK has a role in the stimulation of glycolysis during ischaemia

Trimetazidine improves left ventricular function in diabetic patients with coronary artery disease: a double-blind placebo-controlled study.

Mechanisms controlling mitochondrial biogenesis and respiration through the thermogenic coactivator PGC-1

Perhexiline maleate and peripheral neuropathy

Importance of free fatty acids as a determinant of myocardial oxygen consumption and myocardial ischemic injury during norepinephrine infusion in dogs

The antianginal drug trimetazidine shifts cardiac energy metabolism from fatty acid oxidation to glucose oxidation by inhibiting mitochondrial long-chain 3-ketoacyl coenzyme A thiolase

Long term cardioprotective action of trimetazidine and potential effect on the inflammatory process in patients with ischaemic dilated cardiomyopathy.

Haemodynamic effects of intracoronary pyruvate in patients with congestive heart failure: an open study.

Effect of free fatty acids on myocardial function and oxygen consumption in intact dogs

Defective lipid metabolism in the failing heart

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525-535

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scholarly article

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10.2217/14796678.3.5.525

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19804308

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