Contribution of canonical nonhomologous end joining to chromosomal rearrangements is enhanced by ATM kinase deficiency. - Wikidata - awgldk - TriplyDB

Contribution of canonical nonhomologous end joining to chromosomal rearrangements is enhanced by ATM kinase deficiency.

Contribution of canonical nonhomologous end joining to chromosomal rearrangements is enhanced by ATM kinase deficiency.

http://www.wikidata.org/entity/Q37612501

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C-NHEJ without indels is robust and requires synergistic function of distinct XLF domains.Repeat-mediated deletions can be induced by a chromosomal break far from a repeat, but multiple pathways suppress such rearrangements

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Contribution of canonical nonhomologous end joining to chromosomal rearrangements is enhanced by ATM kinase deficiency.

http://www.wikidata.org/entity/Q37612501

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article científic

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article scientifique

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articolo scientifico

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bilimsel makale

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scientific article published on 05 January 2017

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vedecký článok

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vetenskaplig artikel

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Contribution of canonical nonh ...... nced by ATM kinase deficiency.

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Contribution of canonical nonh ...... nced by ATM kinase deficiency.

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Contribution of canonical nonh ...... nced by ATM kinase deficiency.

@en

Contribution of canonical nonh ...... nced by ATM kinase deficiency.

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Contribution of canonical nonh ...... nced by ATM kinase deficiency.

@en

Contribution of canonical nonh ...... nced by ATM kinase deficiency.

@nl

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PNAS.1612204114

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Proceedings of the National Academy of Sciences of the United States of America

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Contribution of canonical nonh ...... nced by ATM kinase deficiency.

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Caree R Carson

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Gabriella Lee

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Jeremy M Stark

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Ragini Bhargava

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P2860

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Complex landscapes of somatic rearrangement in human breast cancer genomes.

ATM phosphorylates histone H2AX in response to DNA double-strand breaks

Ku70-deficient embryonic stem cells have increased ionizing radiosensitivity, defective DNA end-binding activity, and inability to support V(D)J recombination

A critical role for DNA end-joining proteins in both lymphogenesis and neurogenesis

ATM limits incorrect end utilization during non-homologous end joining of multiple chromosome breaks

Genomic instability in mice lacking histone H2AX

Role of 53BP1 oligomerization in regulating double-strand break repair.

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scholarly article

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10.1073/PNAS.1612204114

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114

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2017-01-05T00:00:00Z

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