Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation. - Wikidata - awgldk - TriplyDB

Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

http://www.wikidata.org/entity/Q37612535

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Auditory cortex interneuron development requires cadherins operating hair-cell mechanoelectrical transduction Large-Scale Cognitive GWAS Meta-Analysis Reveals Tissue-Specific Neural Expression and Potential Nootropic Drug Targets.Amyloid β synaptotoxicity is Wnt-PCP dependent and blocked by fasudil.Vangl2 regulates spermatid planar cell polarity through microtubule (MT)-based cytoskeleton in the rat testis.Wnts Promote Synaptic Assembly Through T-Cell Specific Transcription Factors in Caenorhabditis elegans.A role for APP in Wnt signalling links synapse loss with β-amyloid production

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Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

http://www.wikidata.org/entity/Q37612535

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 05 January 2017

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

@en

Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

@nl

type

label

Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

@en

Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

@nl

prefLabel

Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

@en

Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

@nl

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PNAS.1612062114

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Proceedings of the National Academy of Sciences of the United States of America

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Evidence for opposing roles of Celsr3 and Vangl2 in glutamatergic synapse formation.

@en

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Catarina Fernandes

http://www.w3.org/2001/XMLSchema#string

Darwin K Berg

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Jiaxuan Qi

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John R Yates

http://www.w3.org/2001/XMLSchema#string

John Scott

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Keisuke Onishi

http://www.w3.org/2001/XMLSchema#string

Liqing Wang

http://www.w3.org/2001/XMLSchema#string

Mao Ye

http://www.w3.org/2001/XMLSchema#string

Sonal Thakar

http://www.w3.org/2001/XMLSchema#string

Ting Yu

http://www.w3.org/2001/XMLSchema#string

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A homozygous mutation in human PRICKLE1 causes an autosomal-recessive progressive myoclonus epilepsy-ataxia syndrome

Wnts acting through canonical and noncanonical signaling pathways exert opposite effects on hippocampal synapse formation

The structure of postsynaptic densities isolated from dog cerebral cortex. I. Overall morphology and protein composition

The presynaptic active zone

Neuroligin-1 deletion results in impaired spatial memory and increased repetitive behavior.

Axon guidance and injury-lessons from Wnts and Wnt signaling

PRICKLE1 interaction with SYNAPSIN I reveals a role in autism spectrum disorders

Comparison of phenotypes between different vangl2 mutants demonstrates dominant effects of the Looptail mutation during hair cell development

NF-kappa B functions in synaptic signaling and behavior

Protocadherin Celsr3 is crucial in axonal tract development

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E610-E618

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10.1073/PNAS.1612062114

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2017-01-05T00:00:00Z

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