Chronic alcohol disrupts dopamine receptor activity and the cognitive function of the medial prefrontal cortex. - Wikidata - awgldk - TriplyDB

Chronic alcohol disrupts dopamine receptor activity and the cognitive function of the medial prefrontal cortex.

Chronic alcohol disrupts dopamine receptor activity and the cognitive function of the medial prefrontal cortex.

http://www.wikidata.org/entity/Q37617401

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Thinking after Drinking: Impaired Hippocampal-Dependent Cognition in Human Alcoholics and Animal Models of Alcohol Dependence Corticostriatal circuitry and habitual ethanol seeking Adolescent intermittent ethanol exposure is associated with increased risky choice and decreased dopaminergic and cholinergic neuron markers in adult rats.Binge-Like Alcohol Exposure During Adolescence Disrupts Dopaminergic Neurotransmission in the Adult Prelimbic Cortex.Habitual alcohol seeking: modeling the transition from casual drinking to addiction.Ethanol reduces evoked dopamine release and slows clearance in the rat medial prefrontal cortex.Operant procedures for assessing behavioral flexibility in rats.Dendritic remodeling of hippocampal neurons is associated with altered NMDA receptor expression in alcohol dependent rats Brain-derived neurotrophic factor and addiction: Pathological versus therapeutic effects on drug seeking.Brain pathways to recovery from alcohol dependence Failure to Recognize Novelty after Extended Methamphetamine Self-Administration Results from Loss of Long-Term Depression in the Perirhinal Cortex Glutamatergic plasticity and alcohol dependence-induced alterations in reward, affect and cognition.Alcohol-induced alterations in dopamine modulation of prefrontal activity Chronic Intermittent Ethanol Exposure Enhances the Excitability and Synaptic Plasticity of Lateral Orbitofrontal Cortex Neurons and Induces a Tolerance to the Acute Inhibitory Actions of Ethanol.Translational Research on Habit and Alcohol Adolescent binge-like alcohol alters sensitivity to acute alcohol effects on dopamine release in the nucleus accumbens of adult rats Role for the Rostromedial Tegmental Nucleus in Signaling the Aversive Properties of Alcohol."Boomerang Neuropathology" of Late-Onset Alzheimer's Disease is Shrouded in Harmful "BDDS": Breathing, Diet, Drinking, and Sleep During Aging.The Need for Treatment Responsive Translational Biomarkers in Alcoholism Research.Synaptic targets: Chronic alcohol actions.Lycopene Pretreatment Ameliorates Acute Ethanol Induced NAD(+) Depletion in Human Astroglial Cells.Medial Prefrontal Cortical Dopamine Responses During Operant Self-Administration of Sweetened Ethanol.FMRP Mediates Chronic Ethanol-Induced Changes in NMDA, Kv4.2, and KChIP3 Expression in the Hippocampus.Quetiapine mitigates the ethanol-induced oxidative stress in brain tissue, but not in the liver, of the rat The role of the orbitofrontal cortex in alcohol use, abuse, and dependence.Phosphorylation of calcium/calmodulin-dependent protein kinase II in the rat dorsal medial prefrontal cortex is associated with alcohol-induced cognitive inflexibility.Trait and state binge eating predispose towards cocaine craving.Loss of δ-GABAA receptor-mediated tonic currents in the adult prelimbic cortex following adolescent alcohol exposure.Changes in Neuropsychological Status during the Initial Phase of Abstinence in Alcohol Use Disorder: Neurocognitive Impairment and Implications for Clinical Care.Deficits in the extinction of ethanol-seeking behavior following chronic intermittent ethanol exposure are attenuated with positive allosteric modulation of mGlu5.Morphological and functional evidence of increased excitatory signaling in the prelimbic cortex during ethanol withdrawal.Reversal learning and experimenter-administered chronic intermittent ethanol exposure in male rats.Ethanol Dependence Abolishes Monoamine and GIRK (Kir3) Channel Inhibition of Orbitofrontal Cortex Excitability.Effects of monoamines on the intrinsic excitability of lateral orbitofrontal cortex neurons in alcohol-dependent and non-dependent female mice.ProBDNF/p75NTR/sortilin pathway is activated in peripheral blood of patients with alcohol dependence.

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Chronic alcohol disrupts dopamine receptor activity and the cognitive function of the medial prefrontal cortex.

http://www.wikidata.org/entity/Q37617401

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on March 2014

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Chronic alcohol disrupts dopam ...... the medial prefrontal cortex.

@en

Chronic alcohol disrupts dopam ...... the medial prefrontal cortex.

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type

label

Chronic alcohol disrupts dopam ...... the medial prefrontal cortex.

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Chronic alcohol disrupts dopam ...... the medial prefrontal cortex.

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prefLabel

Chronic alcohol disrupts dopam ...... the medial prefrontal cortex.

@en

Chronic alcohol disrupts dopam ...... the medial prefrontal cortex.

@nl

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JNEUROSCI.0623-13.2014

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Journal of Neuroscience

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Chronic alcohol disrupts dopam ...... the medial prefrontal cortex.

@en

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Heather Trantham-Davidson

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Justin T Gass

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L Judson Chandler

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Marcelo F Lopez

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Patrick J Mulholland

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Samuel W Centanni

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P2860

Thalamocortical projections have a K+ channel that is phosphorylated and modulated by cAMP-dependent protein kinase

Regulation of working memory by dopamine D4 receptor in rats

K(+) channel expression distinguishes subpopulations of parvalbumin- and somatostatin-containing neocortical interneurons

Rapid developmental maturation of neocortical FS cell intrinsic excitability.

Association of frontal and posterior cortical gray matter volume with time to alcohol relapse: a prospective study

Drd2 expression in the high alcohol-preferring and low alcohol-preferring mice.

Striatal dopamine d2/d3 receptor availability is reduced in methamphetamine dependence and is linked to impulsivity

Dopamine D₂ and acetylcholine α7 nicotinic receptors have subcellular distributions favoring mediation of convergent signaling in the mouse ventral tegmental area.

Contributions of Kv3 channels to neuronal excitability.

Addiction and its reward process through polymorphisms of the D2 dopamine receptor gene: a review.

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10.1523/JNEUROSCI.0623-13.2014

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10

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34

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Stan B. Floresco

Elizabeth J Glover

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2014-03-01T00:00:00Z

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24599469

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3942586

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