Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.
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IGFBP-4 regulates adult skeletal growth in a sex-specific manner.The inflammatory phase of fracture healing is influenced by oestrogen status in miceThe Contribution of Experimental in vivo Models to Understanding the Mechanisms of Adaptation to Mechanical Loading in BoneSex steroid actions in male boneEstrogens and Androgens in Skeletal Physiology and Pathophysiology.Aryl hydrocarbon receptors in osteoclast lineage cells are a negative regulator of bone mass.Effects of Deletion of ERα in Osteoblast-Lineage Cells on Bone Mass and Adaptation to Mechanical Loading Differ in Female and Male MiceBeta-Catenin Haplo Insufficient Male Mice Do Not Lose Bone in Response to Hindlimb Unloading.Inhibition of Midkine Augments Osteoporotic Fracture Healing.Dynamic interplay between bone and multiple myeloma: emerging roles of the osteoblastDeletion of Estrogen Receptor Beta in Osteoprogenitor Cells Increases Trabecular but Not Cortical Bone Mass in Female Mice.Transcriptional profiling of cortical versus cancellous bone from mechanically-loaded murine tibiae reveals differential gene expression.sFRP4-dependent Wnt signal modulation is critical for bone remodeling during postnatal development and age-related bone lossThe Effects of Androgens on Murine Cortical Bone Do Not Require AR or ERα Signaling in Osteoblasts and OsteoclastsRegulation of Bone Metabolism by Sex Steroids.Estrogen Regulates Bone Turnover by Targeting RANKL Expression in Bone Lining Cells.miR-199a-3p is involved in estrogen-mediated autophagy through the IGF-1/mTOR pathway in osteocyte-like MLO-Y4 cells.The Role of the Osteocyte in Bone and Nonbone Disease.From top to bottom: midkine and pleiotrophin as emerging players in immune regulation.Effect of a single injection of testosterone enanthate on 17β estradiol and bone turnover markers in hypogonadal male patients.Mouse models to evaluate the role of estrogen receptor α in skeletal maintenance and adaptation.Osteoimmunology: The Conceptual Framework Unifying the Immune and Skeletal Systems.RANKL (Receptor Activator of NFκB Ligand) Produced by Osteocytes Is Required for the Increase in B Cells and Bone Loss Caused by Estrogen Deficiency in Mice.Use of Estradiol Promotes Graft-Bone Healing in Rabbit Model of Anterior Cruciate Ligament Reconstruction With a Polyethylene Terephthalate Ligament.The Role of Estrogen Receptor in Bone Cells
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P2860
Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 10 December 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.
@en
Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.
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type
label
Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.
@en
Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.
@nl
prefLabel
Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.
@en
Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.
@nl
P2093
P2860
P1433
P1476
Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.
@en
P2093
Erina Inoue
Hiroe Sugizaki
Jun K Takeuchi
Katsuhide Igarashi
Kazuki Inoue
Lynda F Bonewald
Shino Kondoh
Taiyong Yu
Yuko Shirode-Fukuda
P2860
P356
10.1016/J.BONE.2013.12.005
P577
2013-12-10T00:00:00Z