FGFR3-TACC3 fusion proteins act as naturally occurring drivers of tumor resistance by functionally substituting for EGFR/ERK signaling.
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A novel read-through transcript JMJD7-PLA2G4B regulates head and neck squamous cell carcinoma cell proliferation and survival.FGFR3-TACC3 cancer gene fusions cause mitotic defects by removal of endogenous TACC3 from the mitotic spindleSystematic Identification of Oncogenic EGFR Interaction PartnersUnique signalling connectivity of FGFR3-TACC3 oncoprotein revealed by quantitative phosphoproteomics and differential network analysis.Are Fusion Transcripts in Relapsed/Metastatic Head and Neck Cancer Patients Predictive of Response to Anti-EGFR Therapies?Concomitant driver mutations in advanced EGFR-mutated non-small-cell lung cancer and their impact on erlotinib treatment.Genomic Landscapes of EBV-Associated Nasopharyngeal Carcinoma vs. HPV-Associated Head and Neck CancerOncogenic driver FGFR3-TACC3 is dependent on membrane trafficking and ERK signaling
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FGFR3-TACC3 fusion proteins act as naturally occurring drivers of tumor resistance by functionally substituting for EGFR/ERK signaling.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 27 June 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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FGFR3-TACC3 fusion proteins ac ...... tuting for EGFR/ERK signaling.
@en
FGFR3-TACC3 fusion proteins ac ...... tuting for EGFR/ERK signaling.
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type
label
FGFR3-TACC3 fusion proteins ac ...... tuting for EGFR/ERK signaling.
@en
FGFR3-TACC3 fusion proteins ac ...... tuting for EGFR/ERK signaling.
@nl
prefLabel
FGFR3-TACC3 fusion proteins ac ...... tuting for EGFR/ERK signaling.
@en
FGFR3-TACC3 fusion proteins ac ...... tuting for EGFR/ERK signaling.
@nl
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FGFR3-TACC3 fusion proteins ac ...... tuting for EGFR/ERK signaling.
@en
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C Castanaro
G Thurston
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10.1038/ONC.2016.216
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P577
2016-06-27T00:00:00Z