Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.
about
Elk-1 a transcription factor with multiple facets in the brainJNKK1 organizes a MAP kinase module through specific and sequential interactions with upstream and downstream components mediated by its amino-terminal extensionMarvelD3 couples tight junctions to the MEKK1-JNK pathway to regulate cell behavior and survivalCDC42 and FGD1 cause distinct signaling and transforming activities.Convergence of MAP kinase pathways on the ternary complex factor Sap-1a.BMK1/ERK5 regulates serum-induced early gene expression through transcription factor MEF2CThe net repressor is regulated by nuclear export in response to anisomycin, UV, and heat shockThe tumor promoter arsenite stimulates AP-1 activity by inhibiting a JNK phosphataseFGF and stress regulate CREB and ATF-1 via a pathway involving p38 MAP kinase and MAPKAP kinase-2Purification and cDNA cloning of SAPKK3, the major activator of RK/p38 in stress- and cytokine-stimulated monocytes and epithelial cellsCD95 promotes tumour growthSelective response of ternary complex factor Sap1a to different mitogen-activated protein kinase subgroupsBeta-arrestin-dependent mu-opioid receptor-activated extracellular signal-regulated kinases (ERKs) Translocate to Nucleus in Contrast to G protein-dependent ERK activation3pK, a novel mitogen-activated protein (MAP) kinase-activated protein kinase, is targeted by three MAP kinase pathwaysA comprehensive pathway map of epidermal growth factor receptor signalingImpaired c-Jun amino terminal kinase activity and T cell differentiation in death receptor 6-deficient miceJNK Signaling: Regulation and Functions Based on Complex Protein-Protein PartnershipsHydrogen peroxide sensing, signaling and regulation of transcription factorsThe MKK7 gene encodes a group of c-Jun NH2-terminal kinase kinasesc-Jun N-terminal kinase-interacting protein 1 inhibits gene expression in response to hypertrophic agonists in neonatal rat ventricular myocytesjkk-1 and mek-1 regulate body movement coordination and response to heavy metals through jnk-1 in Caenorhabditis elegansKeratin-dependent, epithelial resistance to tumor necrosis factor-induced apoptosisRegulation of Schistosoma mansoni development and reproduction by the mitogen-activated protein kinase signaling pathwayAlcohol Regulates Genes that Are Associated with Response to Endocrine Therapy and Attenuates the Actions of Tamoxifen in Breast Cancer CellsMAP kinase- and Rho-dependent signals interact to regulate gene expression but not actin morphology in cardiac muscle cellsRsk-2 activity is necessary for epidermal growth factor-induced phosphorylation of CREB protein and transcription of c-fos geneThe adaptor protein Crk connects multiple cellular stimuli to the JNK signaling pathwayActivation of the JNK pathway is essential for transformation by the Met oncogeneMitogen-activated protein kinases: specific messages from ubiquitous messengersBiochemical and biological functions of the N-terminal, noncatalytic domain of extracellular signal-regulated kinase 2.GnRH induces the c-Fos gene via phosphorylation of SRF by the calcium/calmodulin kinase II pathwayRole of p38 and JNK mitogen-activated protein kinases in the activation of ternary complex factorsNovel membrane-targeted ERK1 and ERK2 chimeras which act as dominant negative, isotype-specific mitogen-activated protein kinase inhibitors of Ras-Raf-mediated transcriptional activation of c-fos in NIH 3T3 cells.RETRACTED: Beta-like importins mediate the nuclear translocation of mitogen-activated protein kinasesMouse preimplantation embryo responses to culture medium osmolarity include increased expression of CCM2 and p38 MAPK activationExercise and Glycemic Control: Focus on Redox Homeostasis and Redox-Sensitive Protein Signaling.Sustained c-Jun-NH2-kinase activity promotes epithelial-mesenchymal transition, invasion, and survival of breast cancer cells by regulating extracellular signal-regulated kinase activation.The Elk-1 ETS-domain transcription factor contains a mitogen-activated protein kinase targeting motif.AP-1 factors play an important role in transformation induced by the v-rel oncogene.Novel roles of specific isoforms of protein kinase C in activation of the c-fos serum response element
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P2860
Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 1995
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.
@en
Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.
@nl
type
label
Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.
@en
Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.
@nl
prefLabel
Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.
@en
Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.
@nl
P2093
P2860
P1433
P1476
Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation
@en
P2093
P2860
P304
P356
10.1002/J.1460-2075.1995.TB00284.X
P407
P577
1995-12-01T00:00:00Z