Neuronal extracellular signal-regulated kinase (ERK) activity as marker and mediator of alcohol and opioid dependence.
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Ethanol-Associated Changes in Glutamate Reward Neurocircuitry: A Minireview of Clinical and Preclinical Genetic Findings.Traumatic brain injury induces neuroinflammation and neuronal degeneration that is associated with escalated alcohol self-administration in ratsThe addicted brain: understanding the neurophysiological mechanisms of addictive disorders.Operant ethanol self-administration increases extracellular-signal regulated protein kinase (ERK) phosphorylation in reward-related brain regions: selective regulation of positive reinforcement in the prefrontal cortex of C57BL/6J mice.The affective dimension of pain as a risk factor for drug and alcohol addiction.Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.Neurobiological Correlates of Pain Avoidance-Like Behavior in Morphine-Dependent and Non-Dependent Rats.Genome-wide meta-analysis of copy number variations with alcohol dependence.
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Neuronal extracellular signal-regulated kinase (ERK) activity as marker and mediator of alcohol and opioid dependence.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 11 March 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Neuronal extracellular signal- ...... alcohol and opioid dependence.
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Neuronal extracellular signal-regulated kinase
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type
label
Neuronal extracellular signal- ...... alcohol and opioid dependence.
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Neuronal extracellular signal-regulated kinase
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prefLabel
Neuronal extracellular signal- ...... alcohol and opioid dependence.
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Neuronal extracellular signal-regulated kinase
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P2860
P356
P1476
Neuronal extracellular signal- ...... alcohol and opioid dependence.
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Eva R Zamora-Martinez
Scott Edwards
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P356
10.3389/FNINT.2014.00024
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2014-03-11T00:00:00Z