Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke.
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A Review of the Mechanisms of Blood-Brain Barrier Permeability by Tissue-Type Plasminogen Activator Treatment for Cerebral IschemiaTissue-type plasminogen activator is a neuroprotectant in the central nervous systemThe ischemic environment drives microglia and macrophage functionAdaptive cellular stress pathways as therapeutic targets of dietary phytochemicals: focus on the nervous systemThe HIF-1/glial TIM-3 axis controls inflammation-associated brain damage under hypoxiaHypoxia Induces autophagic cell death through hypoxia-inducible factor 1α in microgliaIschemia induces different levels of hypoxia inducible factor-1α protein expression in interneurons and pyramidal neurons.Relative Contribution of Prolyl Hydroxylase-Dependent and -Independent Degradation of HIF-1alpha by Proteasomal Pathways in Cerebral Ischemia.Endothelial progenitor cells transplantation attenuated blood-brain barrier damage after ischemia in diabetic mice via HIF-1α.Aestivation and hypoxia-related events share common silent neuron trafficking processes.Melatonin combined with exercise cannot alleviate cerebral injury in a rat model of focal cerebral ischemia/reperfusion injury.MiR-335 Regulates Hif-1α to Reduce Cell Death in Both Mouse Cell Line and Rat Ischemic Models.Tissue-type plasminogen activator regulates the neuronal uptake of glucose in the ischemic brain.Dipeptidyl peptidase 4 inhibition may facilitate healing of chronic foot ulcers in patients with type 2 diabetes.Plasma B-type natriuretic peptide level in patients with acute cerebral infarction according to infarction subtype and infarction volumeHinokitiol, a natural tropolone derivative, offers neuroprotection from thromboembolic stroke in vivoEvidence that collaboration between HIF-1α and Notch-1 promotes neuronal cell death in ischemic stroke.A Crucial Role of CXCL14 for Promoting Regulatory T Cells Activation in StrokeRole of transcription factors in neurogenesis after cerebral ischemia.Epigenetics: new questions on the response to hypoxia.Hypoxia inducible factor-1 as a target for neurodegenerative diseases.Pleiotropic effects of dronedarone on ischemia/reperfusion injury in heart and brain.Robustness of the hypoxic response: another job for miRNAs?The novel proteasome inhibitor BSc2118 protects against cerebral ischaemia through HIF1A accumulation and enhanced angioneurogenesis.SIRT1 regulation modulates stroke outcome.Chinese medicines and bioactive compounds for treatment of stroke.Perinatal Brain Injury As a Consequence of Preterm Birth and Intrauterine Inflammation: Designing Targeted Stem Cell Therapies.A Genetically Encoded FRET Sensor for Hypoxia and Prolyl Hydroxylases.Angiogenesis, neurogenesis and neuroplasticity in ischemic stroke.β2-Adrenergic Receptor-Mediated HIF-1α Upregulation Mediates Blood Brain Barrier Damage in Acute Cerebral Ischemia.Treatment with an activator of hypoxia-inducible factor 1, DMOG provides neuroprotection after traumatic brain injury.Neuroprotection against cobalt chloride-induced cell apoptosis of primary cultured cortical neurons by salidroside.HIF-1α involves in neuronal apoptosis after traumatic brain injury in adult rats.Neuronal Rho GTPase Rac1 elimination confers neuroprotection in a mouse model of permanent ischemic stroke.HIF-1α triggers long-lasting glutamate excitotoxicity via system xc- in cerebral ischaemia-reperfusion.The early activation of PI3K strongly enhances the resistance of cortical neurons to hypoxic injury via the activation of downstream targets of the PI3K pathway and the normalization of the levels of PARP activity, ATP, and NAD⁺.The effect of one night of continuous positive airway pressure therapy on oxidative stress and antioxidant defense in hypertensive patients with severe obstructive sleep apnea.Systemic Proteasome Inhibition Induces Sustained Post-stroke Neurological Recovery and Neuroprotection via Mechanisms Involving Reversal of Peripheral Immunosuppression and Preservation of Blood-Brain-Barrier Integrity.Potential advantages of Chinese medicine Taohong Siwu Decoction () combined with tissue-plasminogen activator for alleviating middle cerebral artery occlusion-induced embolic stroke in rats.Chronodisruption increases cardiovascular risk in zebrafish via reduced clearance of senescent erythrocytes.
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Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
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name
Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke.
@en
Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke.
@nl
type
label
Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke.
@en
Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke.
@nl
prefLabel
Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke.
@en
Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke.
@nl
P2860
P1476
Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke.
@en
P2093
Honglian Shi
P2860
P304
P356
10.2174/092986709789760779
P577
2009-01-01T00:00:00Z