Bidirectional regulation of P body formation mediated by eIF4F complex formation in sensory neurons.
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Commonalities between pain and memory mechanisms and their meaning for understanding chronic painThe anti-diabetic drug metformin protects against chemotherapy-induced peripheral neuropathy in a mouse modelAdenosine Monophosphate-activated Protein Kinase (AMPK) Activators For the Prevention, Treatment and Potential Reversal of Pathological Pain.Aberrant plasticity of peripheral sensory axons in a painful neuropathyLocal translation and retrograde axonal transport of CREB regulates IL-6-induced nociceptive plasticity.The pharmacology of nociceptor primingRNA transport and long-term memory storage.The potent, indirect adenosine monophosphate- activated protein kinase activator R419 attenuates mitogen-activated protein kinase signaling, inhibits nociceptor excitability, and reduces pain hypersensitivity in mice.
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P2860
Bidirectional regulation of P body formation mediated by eIF4F complex formation in sensory neurons.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 27 September 2013
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Bidirectional regulation of P ...... formation in sensory neurons.
@en
Bidirectional regulation of P ...... formation in sensory neurons.
@nl
type
label
Bidirectional regulation of P ...... formation in sensory neurons.
@en
Bidirectional regulation of P ...... formation in sensory neurons.
@nl
prefLabel
Bidirectional regulation of P ...... formation in sensory neurons.
@en
Bidirectional regulation of P ...... formation in sensory neurons.
@nl
P2093
P2860
P1433
P1476
Bidirectional regulation of P ...... formation in sensory neurons.
@en
P2093
Galo L Mejia
Ohannes K Melemedjian
Olivia K Zoph
Talya S Lepow
P2860
P304
P356
10.1016/J.NEULET.2013.09.048
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P577
2013-09-27T00:00:00Z