Progress and perspective of TBX6 gene in congenital vertebral malformations. - Wikidata - awgldk - TriplyDB

Progress and perspective of TBX6 gene in congenital vertebral malformations.

Progress and perspective of TBX6 gene in congenital vertebral malformations.

http://www.wikidata.org/entity/Q37636813

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Progress and Application of CRISPR/Cas Technology in Biological and Biomedical Investigation.Comparative analysis of serum proteome in congenital scoliosis patients with TBX6 haploinsufficiency - a first report pointing to lipid metabolism.

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Progress and perspective of TBX6 gene in congenital vertebral malformations.

http://www.wikidata.org/entity/Q37636813

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 15 July 2016

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Progress and perspective of TBX6 gene in congenital vertebral malformations.

@en

Progress and perspective of TBX6 gene in congenital vertebral malformations.

@nl

type

label

Progress and perspective of TBX6 gene in congenital vertebral malformations.

@en

Progress and perspective of TBX6 gene in congenital vertebral malformations.

@nl

prefLabel

Progress and perspective of TBX6 gene in congenital vertebral malformations.

@en

Progress and perspective of TBX6 gene in congenital vertebral malformations.

@nl

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ONCOTARGET.10619

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Progress and perspective of TBX6 gene in congenital vertebral malformations.

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Dongtang Yuan

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Jiaqi Liu

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Philip F Giampietro

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Qiankun Zhu

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Sen Liu

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Shishu Huang

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Weisheng Chen

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Yuzhi Zuo

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Zhenlei Liu

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Zhihong Wu

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Creative Commons Attribution 2.5 Unported

P2860

Mutations in the MESP2 gene cause spondylothoracic dysostosis/Jarcho-Levin syndrome

The human TBX6 gene: cloning and assignment to chromosome 16p11.2

Mutations in the human delta homologue, DLL3, cause axial skeletal defects in spondylocostal dysostosis

Mutated MESP2 causes spondylocostal dysostosis in humans.

Mutation of the LUNATIC FRINGE gene in humans causes spondylocostal dysostosis with a severe vertebral phenotype

Control of Hes7 expression by Tbx6, the Wnt pathway and the chemical Gsk3 inhibitor LiCl in the mouse segmentation clock

Axial skeletal defects caused by mutation in the spondylocostal dysplasia/pudgy gene Dll3 are associated with disruption of the segmentation clock within the presomitic mesoderm

Overlapping functions of Pea3 ETS transcription factors in FGF signaling during zebrafish development

Instability of Hes7 protein is crucial for the somite segmentation clock

The Mesp2 transcription factor establishes segmental borders by suppressing Notch activity

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10.18632/ONCOTARGET.10619

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2016-08-30T00:00:00Z

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