Myocardial remodeling after infarction: the role of myofibroblasts.
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How Biomaterials Can Influence Various Cell Types in the Repair and Regeneration of the Heart after Myocardial InfarctionAntifibrotic therapies to control cardiac fibrosisAtrial Fibrillation and Fibrosis: Beyond the Cardiomyocyte Centric ViewRecent developments in myofibroblast biology: paradigms for connective tissue remodelingThe role of the myofibroblast in tumor stroma remodelingHydrogen Sulfide as a Potential Therapeutic Target in FibrosisMMI-0100 inhibits cardiac fibrosis in myocardial infarction by direct actions on cardiomyocytes and fibroblasts via MK2 inhibition.Cardiac myocyte diversity and a fibroblast network in the junctional region of the zebrafish heart revealed by transmission and serial block-face scanning electron microscopyHypothyroidism and its rapid correction alter cardiac remodelingRegenerating functional heart tissue for myocardial repairCardiac fibrosis in myocardial infarction-from repair and remodeling to regenerationCollagen XIV is important for growth and structural integrity of the myocardiumAn anthelmintic drug, pyrvinium pamoate, thwarts fibrosis and ameliorates myocardial contractile dysfunction in a mouse model of myocardial infarctionTGF-β1-mediated differentiation of fibroblasts is associated with increased mitochondrial content and cellular respirationUridine triphosphate (UTP) induces profibrotic responses in cardiac fibroblasts by activation of P2Y2 receptorsCyclic nucleotide phosphodiesterase 1A: a key regulator of cardiac fibroblast activation and extracellular matrix remodeling in the heartThe constant beat: cardiomyocytes adapt their forces by equal contraction upon environmental stiffeningDirect intercellular communications dominate the interaction between adipose-derived MSCs and myofibroblasts against cardiac fibrosis.Local Gradients in Electrotonic Loading Modulate the Local Effective Refractory Period: Implications for Arrhythmogenesis in the Infarct Border Zone.Cardiac fibroblast in development and wound healing.Integrins αvβ5 and αvβ3 promote latent TGF-β1 activation by human cardiac fibroblast contractionDirect reprogramming of fibroblasts into cardiomyocytesThe Soft- and Hard-Heartedness of Cardiac Fibroblasts: Mechanotransduction Signaling Pathways in Fibrosis of the Heart.Sphingosine 1-Phosphate Receptors: Do They Have a Therapeutic Potential in Cardiac Fibrosis?Cardioprotection against Heart Failure by Shenfu Injection via TGF-β/Smads Signaling Pathway.Exploring the elasticity and adhesion behavior of cardiac fibroblasts by atomic force microscopy indentation.The zebrafish heart regenerates after cryoinjury-induced myocardial infarction.HDAC class I inhibitor, Mocetinostat, reverses cardiac fibrosis in heart failure and diminishes CD90+ cardiac myofibroblast activationRole of adenosine A2B receptor signaling in contribution of cardiac mesenchymal stem-like cells to myocardial scar formation.Progression of diffuse myocardial fibrosis assessed by cardiac magnetic resonance T₁ mapping.Resident cardiac immune cells and expression of the ectonucleotidase enzymes CD39 and CD73 after ischemic injury.Angiotensin converting enzyme (ACE) and ACE2 bind integrins and ACE2 regulates integrin signalling.Mesenchymal stromal cells but not cardiac fibroblasts exert beneficial systemic immunomodulatory effects in experimental myocarditis.Genetically manipulated progenitor cell sheet with diprotin A improves myocardial function and repair of infarcted hearts.Quantitative analysis of cardiac tissue including fibroblasts using three-dimensional confocal microscopy and image reconstruction: towards a basis for electrophysiological modeling.Somatic cell plasticity and Niemann-Pick type C2 protein: fibroblast activation.Disruption of type 5 adenylyl cyclase prevents β-adrenergic receptor cardiomyopathy: a novel approach to β-adrenergic receptor blockade.Novel therapeutic strategies targeting fibroblasts and fibrosis in heart disease.Inhibitory effects of C-type natriuretic peptide on the differentiation of cardiac fibroblasts, and secretion of monocyte chemoattractant protein-1 and plasminogen activator inhibitor-1.Toll-like receptor 9 prevents cardiac rupture after myocardial infarction in mice independently of inflammation.
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P2860
Myocardial remodeling after infarction: the role of myofibroblasts.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Myocardial remodeling after infarction: the role of myofibroblasts.
@en
Myocardial remodeling after infarction: the role of myofibroblasts.
@nl
type
label
Myocardial remodeling after infarction: the role of myofibroblasts.
@en
Myocardial remodeling after infarction: the role of myofibroblasts.
@nl
prefLabel
Myocardial remodeling after infarction: the role of myofibroblasts.
@en
Myocardial remodeling after infarction: the role of myofibroblasts.
@nl
P2093
P1476
Myocardial remodeling after infarction: the role of myofibroblasts.
@en
P2093
Javier Diez
Johan Verjans
Leo Hofstra
Susanne W M van den Borne
W Matthijs Blankesteijn
P356
10.1038/NRCARDIO.2009.199
P407
P50
P577
2009-12-01T00:00:00Z