Liver X receptor α mediates hepatic triglyceride accumulation through upregulation of G0/G1 Switch Gene 2 expression. - Wikidata - awgldk - TriplyDB

Liver X receptor α mediates hepatic triglyceride accumulation through upregulation of G0/G1 Switch Gene 2 expression.

Liver X receptor α mediates hepatic triglyceride accumulation through upregulation of G0/G1 Switch Gene 2 expression.

http://www.wikidata.org/entity/Q37646873

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G0S2: A small giant controller of lipolysis and adipose-liver fatty acid flux.LC3-Associated Phagocytosis and Inflammation.

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Liver X receptor α mediates hepatic triglyceride accumulation through upregulation of G0/G1 Switch Gene 2 expression.

http://www.wikidata.org/entity/Q37646873

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 23 February 2017

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Liver X receptor α mediates he ...... 0/G1 Switch Gene 2 expression.

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Liver X receptor α mediates he ...... 0/G1 Switch Gene 2 expression.

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type

label

Liver X receptor α mediates he ...... 0/G1 Switch Gene 2 expression.

@en

Liver X receptor α mediates he ...... 0/G1 Switch Gene 2 expression.

@nl

prefLabel

Liver X receptor α mediates he ...... 0/G1 Switch Gene 2 expression.

@en

Liver X receptor α mediates he ...... 0/G1 Switch Gene 2 expression.

@nl

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Liver X receptor α mediates he ...... 0/G1 Switch Gene 2 expression.

@en

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Alicia M Saarinen

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Erin E Kershaw

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Jun Liu

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Rudolf Zechner

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Xiaodong Zhang

http://www.w3.org/2001/XMLSchema#string

P2860

Identification, cloning, expression, and purification of three novel human calcium-independent phospholipase A2 family members possessing triacylglycerol lipase and acylglycerol transacylase activities

Identification of liver X receptor-retinoid X receptor as an activator of the sterol regulatory element-binding protein 1c gene promoter

Regulation of mouse sterol regulatory element-binding protein-1c gene (SREBP-1c) by oxysterol receptors, LXRalpha and LXRbeta

Direct and indirect mechanisms for regulation of fatty acid synthase gene expression by liver X receptors

A targeted apolipoprotein B-38.9-producing mutation causes fatty livers in mice due to the reduced ability of apolipoprotein B-38.9 to transport triglycerides

The liver X receptor (LXR) and hepatic lipogenesis. The carbohydrate-response element-binding protein is a target gene of LXR

Liver X receptors as insulin-mediating factors in fatty acid and cholesterol biosynthesis

Gene transcription of the retinoid X receptor alpha (RXRalpha) is regulated by fatty acids and hormones in rat hepatic cells

Liver X receptor (LXR)-beta regulation in LXRalpha-deficient mice: implications for therapeutic targeting

Non-redundant roles for LXRalpha and LXRbeta in atherosclerosis susceptibility in low density lipoprotein receptor knockout mice.

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e88735

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scholarly article

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10.1172/JCI.INSIGHT.88735

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4

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Bradlee L Heckmann

Gabriele Schoiswohl

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2017-02-23T00:00:00Z

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