IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development.
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Loss of TAB3 expression by shRNA exhibits suppressive bioactivity and increased chemical sensitivity of ovarian cancer cell lines via the NF-κB pathway.IKKβ Activation in the Fetal Lung Mesenchyme Alters Lung Vascular Development but Not Airway Morphogenesis.Recent advances in our understanding of the mechanisms of late lung development and bronchopulmonary dysplasia.Inhibition of IκBβ/NFκB signaling prevents LPS-induced IL1β expression without increasing apoptosis in the developing mouse lung.FGF10 and Human Lung Disease Across the Life Spectrum
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IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 07 March 2016
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development.
@en
IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development.
@nl
type
label
IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development.
@en
IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development.
@nl
prefLabel
IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development.
@en
IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development.
@nl
P2093
P2860
P356
P1476
IL-1β and Inflammasome Activity Link Inflammation to Abnormal Fetal Airway Development
@en
P2093
Alyssa M McCoy
Ashley N Stouch
Hal M Hoffman
Lawrence S Prince
Omar Lakhdari
Rachel M Greer
Timothy S Blackwell
P2860
P304
P356
10.4049/JIMMUNOL.1500906
P407
P577
2016-03-07T00:00:00Z