APC resistance: biological basis and acquired influences. - Wikidata - awgldk - TriplyDB

APC resistance: biological basis and acquired influences.

APC resistance: biological basis and acquired influences.

http://www.wikidata.org/entity/Q37651037

about

Combined oral contraceptives: venous thrombosis Combined oral contraceptives: venous thrombosis Activated protein C anticoagulant system dysfunction and thrombophilia in Asia Protein C anticoagulant and cytoprotective pathways Survival advantage of heterozygous factor V Leiden carriers in murine sepsis.Polymorphisms at activated protein C cleavage sites of factor V: Are they important in the absence of factor V Leiden?Impact of hormone-associated resistance to activated protein C on the thrombotic potential of oral contraceptives: a prospective observational study.Unmasking Asian thrombophilia: is APC dysfunction the real culprit?Prothrombotic risk factors and antithrombotic therapy in children with ischemic stroke Painless Livedoid Vasculopathy in a Patient with G20210A Prothrombin Gene Mutation Coagulation factor V mediates inhibition of tissue factor signaling by activated protein C in mice Mechanisms of anticoagulant and cytoprotective actions of the protein C pathway.The phenotypic and genetic assessment of protein C deficiency.Current knowledge on the genetics of incident venous thrombosis.Primary Thrombophilia in México XI: Activated Protein C Resistance Phenotypes are Multifactorial.Novel insights into the regulation of coagulation by factor V isoforms, tissue factor pathway inhibitorα, and protein S.Diagnosis and management of factor V Leiden.Genetic Risk Factors in Venous Thromboembolism.Extracellular Matrix Reorganization During Wound Healing and Its Impact on Abnormal Scarring.Evaluation of role of FV, FVIII and APLAs in the pathogenesis of APCR in FV Leiden negative DVT patients: a study in India.Plasma hypercoagulability in the presence of thrombomodulin but not of activated protein C in patients with cirrhosis.Identification and functional characterization of a novel F5 mutation (Ala512Val, FVB onn ) associated with activated protein C resistance.Maximising the diagnostic potential of APTT-based screening assays for activated protein C resistance.Prevalence of Factor V Genetic Variants Associated With Indian APCR Contributing to Thrombotic Risk.Pathology Consultation on the Laboratory Evaluation of Thrombophilia Genome wide association study for plasma levels of natural anticoagulant inhibitors and protein C anticoagulant pathway: the MARTHA project

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P2860

APC resistance: biological basis and acquired influences.

http://www.wikidata.org/entity/Q37651037

description

2009 nî lūn-bûn

@nan

2009年の論文

@ja

2009年学术文章

@wuu

2009年学术文章

@zh-cn

2009年学术文章

@zh-hans

2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

2009年學術文章

@zh

2009年學術文章

@zh-hant

name

APC resistance: biological basis and acquired influences.

@en

APC resistance: biological basis and acquired influences.

@nl

type

label

APC resistance: biological basis and acquired influences.

@en

APC resistance: biological basis and acquired influences.

@nl

prefLabel

APC resistance: biological basis and acquired influences.

@en

APC resistance: biological basis and acquired influences.

@nl

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P356

J.1538-7836.2009.03711.X

P1433

Journal of Thrombosis and Haemostasis

P1476

APC resistance: biological basis and acquired influences.

@en

P2093

E Castoldi

http://www.w3.org/2001/XMLSchema#string

J Rosing

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P2860

Familial thrombophilia due to a previously unrecognized mechanism characterized by poor anticoagulant response to activated protein C: prediction of a cofactor to activated protein C

Low-dose oral contraceptives and acquired resistance to activated protein C: a randomised cross-over study

Effects of protein S and factor Xa on peptide bond cleavages during inactivation of factor Va and factor VaR506Q by activated protein C

Peptide bond cleavages and loss of functional activity during inactivation of factor Va and factor VaR506Q by activated protein C.

Mutation in blood coagulation factor V associated with resistance to activated protein C.

The C-terminal region of the factor V B-domain is crucial for the anticoagulant activity of factor V.

Protein S stimulates inhibition of the tissue factor pathway by tissue factor pathway inhibitor.

Inherited resistance to activated protein C is corrected by anticoagulant cofactor activity found to be a property of factor V.

Identification of the same factor V gene mutation in 47 out of 50 thrombosis-prone families with inherited resistance to activated protein C

The anticoagulant protein C pathway.

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445-453

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scholarly article

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10.1111/J.1538-7836.2009.03711.X

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3

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8

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2009-11-30T00:00:00Z

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40680422

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20002539

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