Functional Redundancy of Septin Homologs in Dendritic Branching. - Wikidata - awgldk - TriplyDB

Functional Redundancy of Septin Homologs in Dendritic Branching.

Functional Redundancy of Septin Homologs in Dendritic Branching.

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Functional Redundancy of Septin Homologs in Dendritic Branching.

http://www.wikidata.org/entity/Q37651643

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 20 February 2017

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Functional Redundancy of Septin Homologs in Dendritic Branching.

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Functional Redundancy of Septin Homologs in Dendritic Branching.

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Functional Redundancy of Septin Homologs in Dendritic Branching.

@en

Functional Redundancy of Septin Homologs in Dendritic Branching.

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Functional Redundancy of Septin Homologs in Dendritic Branching.

@en

Functional Redundancy of Septin Homologs in Dendritic Branching.

@nl

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FCELL.2017.00011

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Frontiers in cell and developmental biology

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Functional Redundancy of Septin Homologs in Dendritic Branching

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Charlotte Kaplan

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Mayra Steinmann

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Natalia A Zapiorkowska

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P2860

Analysis of septins across kingdoms reveals orthology and new motifs

Septin1, a new interaction partner for human serine/threonine kinase aurora-B

Mammalian septins nomenclature.

Genetic deletion of SEPT7 reveals a cell type-specific role of septins in microtubule destabilization for the completion of cytokinesis

A Septin-Dependent Diffusion Barrier at Dendritic Spine Necks

Structural insight into filament formation by mammalian septins

Genetic control of the cell division cycle in yeast. IV. Genes controlling bud emergence and cytokinesis.

Septin1 is required for spindle assembly and chromosome congression in mouse oocytes

Disruption of Sept6, a fusion partner gene of MLL, does not affect ontogeny, leukemogenesis induced by MLL-SEPT6, or phenotype induced by the loss of Sept4.

The septin CDCrel-1 is dispensable for normal development and neurotransmitter release

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11

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scholarly article

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10.3389/FCELL.2017.00011

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5

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2017-02-20T00:00:00Z

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28265560

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5316521

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