Methamphetamine self-administration acutely decreases monoaminergic transporter function. - Wikidata - awgldk - TriplyDB

Methamphetamine self-administration acutely decreases monoaminergic transporter function.

Methamphetamine self-administration acutely decreases monoaminergic transporter function.

http://www.wikidata.org/entity/Q37656152

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Mechanisms of dopamine transporter regulation in normal and disease states VMAT1 deletion causes neuronal loss in the hippocampus and neurocognitive deficits in spatial discrimination Chronic wheel running-induced reduction of extinction and reinstatement of methamphetamine seeking in methamphetamine dependent rats is associated with reduced number of periaqueductal gray dopamine neurons.Chronic wheel running reduces maladaptive patterns of methamphetamine intake: regulation by attenuation of methamphetamine-induced neuronal nitric oxide synthase CREB phosphorylation regulates striatal transcriptional responses in the self-administration model of methamphetamine addiction in the rat.Neuroprotection of resveratrol against neurotoxicity induced by methamphetamine in mouse mesencephalic dopaminergic neurons.Metabolites Alterations in the Medial Prefrontal Cortex of Methamphetamine Users in Abstinence: A H MRS Study

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Methamphetamine self-administration acutely decreases monoaminergic transporter function.

http://www.wikidata.org/entity/Q37656152

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 13 December 2011

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

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name

Methamphetamine self-administration acutely decreases monoaminergic transporter function.

@en

Methamphetamine self-administration acutely decreases monoaminergic transporter function.

@nl

type

label

Methamphetamine self-administration acutely decreases monoaminergic transporter function.

@en

Methamphetamine self-administration acutely decreases monoaminergic transporter function.

@nl

prefLabel

Methamphetamine self-administration acutely decreases monoaminergic transporter function.

@en

Methamphetamine self-administration acutely decreases monoaminergic transporter function.

@nl

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Methamphetamine self-administration acutely decreases monoaminergic transporter function

@en

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Annette E Fleckenstein

http://www.w3.org/2001/XMLSchema#string

Diana G Wilkins

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Glen R Hanson

http://www.w3.org/2001/XMLSchema#string

Lisa M McFadden

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Paula L Vieira-Brock

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Scott C Allen

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Shannon M Nielsen

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P2860

4-Methylmethcathinone (mephedrone): neuropharmacological effects of a designer stimulant of abuse

Methamphetamine toxicity and messengers of death

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Regulation of the vesicular monoamine transporter-2: a novel mechanism for cocaine and other psychostimulants

Mechanisms of neurotransmitter release by amphetamines: a review

Methamphetamine self-administration is associated with persistent biochemical alterations in striatal and cortical dopaminergic terminals in the rat

Methamphetamine treatment during development attenuates the dopaminergic deficits caused by subsequent high-dose methamphetamine administration.

Loss of object recognition memory produced by extended access to methamphetamine self-administration is reversed by positive allosteric modulation of metabotropic glutamate receptor 5

Age-dependent differential responses of monoaminergic systems to high doses of methamphetamine.

Methamphetamine-induced rapid and reversible changes in dopamine transporter function: an in vitro model.

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240-245

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scholarly article

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10.1002/SYN.21506

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3

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66

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Kristen A Stout

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2011-12-13T00:00:00Z

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51837723

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22120988

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3962658

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