Cytokine responses against periodontal infection: protective and destructive roles.
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The capsule of Porphyromonas gingivalis leads to a reduction in the host inflammatory response, evasion of phagocytosis, and increase in virulenceThe link between periodontal disease and rheumatoid arthritis: an updated review.Variants in the interleukin-1 alpha and beta genes, and the risk for periodontal disease in dogs.Baicalin downregulates Porphyromonas gingivalis lipopolysaccharide-upregulated IL-6 and IL-8 expression in human oral keratinocytes by negative regulation of TLR signaling.The Kampo medicine Rokumigan possesses antibiofilm, anti-inflammatory, and wound healing properties.Effect of laser on TNF-alpha expression in inflamed human gingival tissue.Oral microbial biofilm stimulation of epithelial cell responses.Mast cells act as phagocytes against the periodontopathogen Aggregatibacter actinomycetemcomitans.Role of Porphyromonas gingivalis phosphoserine phosphatase enzyme SerB in inflammation, immune response, and induction of alveolar bone resorption in ratsTranscriptional profiling of bone marrow stromal cells in response to Porphyromonas gingivalis secreted productsAggressive periodontitis defined by recursive partitioning analysis of immunologic factors.Polymicrobial infection with periodontal pathogens specifically enhances microRNA miR-146a in ApoE-/- mice during experimental periodontal disease.Role of periodontal pathogenic bacteria in RANKL-mediated bone destruction in periodontal disease.NOD2 contributes to Porphyromonas gingivalis-induced bone resorption.The newly discovered cytokine IL-34 is expressed in gingival fibroblasts, shows enhanced expression by pro-inflammatory cytokines, and stimulates osteoclast differentiation.Pro-inflammatory, Th1, Th2, Th17 cytokines and dendritic cells: a cross-sectional study in chronic periodontitis.Effects of the antimicrobial peptide cathelicidin (LL-37) on immortalized gingival fibroblasts infected with Porphyromonas gingivalis and irradiated with 625-nm LED light.Black Tea Extract and Its Theaflavin Derivatives Inhibit the Growth of Periodontopathogens and Modulate Interleukin-8 and β-Defensin Secretion in Oral Epithelial Cells.Comparative analysis of blood and saliva expression profiles in chronic and refractory periodontitis patients.The Daiokanzoto (TJ-84) Kampo Formulation Reduces Virulence Factor Gene Expression in Porphyromonas gingivalis and Possesses Anti-Inflammatory and Anti-Protease Activities.Porphyromonas gingivalis Stimulates Bone Resorption by Enhancing RANKL (Receptor Activator of NF-κB Ligand) through Activation of Toll-like Receptor 2 in Osteoblasts.The Influence of Interleukin 17A and IL17F Polymorphisms on Chronic Periodontitis Disease in Brazilian Patients.Oral inflammatory diseases and systemic inflammation: role of the macrophage.The effect of low-level laser therapy on non-surgical periodontal treatment: a randomized controlled, single-blind, split-mouth clinical trial.MicroRNA-138 Inhibits Periodontal Progenitor Differentiation under Inflammatory ConditionsRole of the NK cell-activating receptor CRACC in periodontitis.Peripheral Blood Leukocytes Interleukin-1 Beta (IL-1β) Cytokine Hyper-Reactivity in Chronic PeriodontitisAntimicrobial and Attractant Roles for Chemerin in the Oral Cavity during Inflammatory Gum DiseaseHow has research into cytokine interactions and their role in driving immune responses impacted our understanding of periodontitis?What can the periodontal community learn from the pathophysiology of rheumatoid arthritis?The RANKL-OPG system in clinical periodontology.The receptor activator of NF-κB ligand-osteoprotegerin system in pulpal and periapical disease.Inflammatory and immune pathways in the pathogenesis of periodontal disease.Role of periodontal ligament fibroblasts in osteoclastogenesis: a review.The complement system and its role in the pathogenesis of periodontitis: current concepts.Enamel matrix derivative, inflammation and soft tissue wound healing.Retinoids stimulate periosteal bone resorption by enhancing the protein RANKL, a response inhibited by monomeric glucocorticoid receptor.Periodontal herpesviruses: prevalence, pathogenicity, systemic risk.Bee Venom Inhibits Porphyromonas gingivalis Lipopolysaccharides-Induced Pro-Inflammatory Cytokines through Suppression of NF-κB and AP-1 Signaling Pathways.Black tea theaflavins attenuate Porphyromonas gingivalis virulence properties, modulate gingival keratinocyte tight junction integrity and exert anti-inflammatory activity.
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P2860
Cytokine responses against periodontal infection: protective and destructive roles.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Cytokine responses against periodontal infection: protective and destructive roles.
@en
Cytokine responses against periodontal infection: protective and destructive roles.
@nl
type
label
Cytokine responses against periodontal infection: protective and destructive roles.
@en
Cytokine responses against periodontal infection: protective and destructive roles.
@nl
prefLabel
Cytokine responses against periodontal infection: protective and destructive roles.
@en
Cytokine responses against periodontal infection: protective and destructive roles.
@nl
P2093
P2860
P1433
P1476
Cytokine responses against periodontal infection: protective and destructive roles.
@en
P2093
Ulf H Lerner
Yen-Chun G Liu
Yen-Tung A Teng
P2860
P304
P356
10.1111/J.1600-0757.2009.00321.X
P577
2010-02-01T00:00:00Z